What Are the Most Common Causes of Mild Chest Pain?

Its basic pathological basis is myocardial ischemia caused by coronary atherosclerosis caused by coronary atherosclerosis and coronary spasm. 1. Inflammation: dermatitis, non-purulent osteochondritis, shingles, myositis, epidemic myalgia (EpidemicMyalgia), pleurisy, pericarditis, mediastinitis, esophagitis, etc.

Chest pain after exhaustion

Coronary heart disease (coronary heart disease is referred to as coronary heart disease.) Due to abnormal lipid metabolism, lipids in the blood are deposited on the original smooth arterial intima, and some atheromatous lipids are accumulated on the intima of the arteries. White plaques are called atherosclerotic lesions. These plaques gradually increase, causing narrowing of the arterial cavity, obstructing blood flow, leading to ischemia of the heart and angina pectoris.) Angina pectoris is the most common cause of chest pain. , Emotional seizures.

Affected area: chest
Related diseases: Coronary heart disease, angina pectoris, sudden death, coronary heart disease, stable angina, variant angina, unstable angina, elderly variant angina, elderly unstable angina, elderly, stable angina, chest pain, chest pain, chest pain

Affiliated Department: Other departments
Related symptoms: Sudden death, atherosclerosis, fatigue, chest pain, myocardial infarction, increased myocardial oxygen consumption, angina pectoris

2. Visceral ischemia: angina pectoris, acute myocardial infarction, cardiomyopathy, pulmonary infarction, etc.

3. Tumor: compression or infiltration of primary lung cancer, mediastinal tumor, myeloma, leukemia, etc.

4. Other reasons: spontaneous pneumothorax, thoracic aortic aneurysm, dissection aneurysm, hyperventilation syndrome, trauma, etc.

5. Cardiac neurosis.

Generally manifested as left chest crushing pain, stuffiness, and sore pain. It often spreads to the back, the inside of the left arm, and the teeth. The duration varies, and generally does not exceed 30 minutes. If after a seizure or after oral nitroglycerin relieves, the diagnosis is basically made. Inflammation of the chest wall skin changes in the skin of the affected area such as redness, swelling, heat, and pain. Shingles is a group of small blisters that are distributed along the nerves and do not cross the midline, with obvious pain. During epidemic myalgia, severe pain in chest and abdomen muscles can occur, which can radiate to shoulders and neck. Non-purulent osteochondritis mostly invades the first and second costal cartilage. The affected area is swollen and painful, but the skin is usually not red and swollen. The pain of angina pectoris and acute myocardial infarction is often located behind the sternum or anterior heart. Pain in esophageal disorders, diaphragmatic hernias, and mediastinal tumors is also located behind the sternum. Spontaneous pneumothorax, acute pleurisy, and pulmonary infarction are often severe chest pains on the affected side. The first task in evaluating chest pain is to distinguish between chest pain in the respiratory system and chest pain associated with other systems, which is not always easy. The nature of the pain and the circumstances in which it occurs can often be used to distinguish pain from angina pectoris or myocardial infarction; it may be difficult to discern pain caused by a mesial aneurysm based on medical history alone.

Differential diagnosis of chest pain after fatigue:

1. Stable angina pectoris: in line with the characteristics of typical angina pectoris, the course of disease lasts 1 month or more. This is because the diseased coronary arteries can only meet the myocardial blood supply in a quiet state or in a lightly tired state. After the increase of labor intensity, myocardial oxygen consumption increased, resulting in transient hypoxia. This type of angina pectoris is characterized by a relatively constant intensity of labor that induces angina pectoris, and symptoms can be relieved after stopping the activity. The pathological basis is often due to the fixed narrowing of the coronary arteries. The course is relatively long and the condition is relatively stable.

2. First-onset overworked angina: Refers to those who meet the typical angina but the course of the disease is within 1 month; those who have not had or have had angina in the past but have not had it for several months. Some of them can be converted to stable labor angina pectoris, but some of them have rapid progress of coronary atherosclerosis, or acute myocardial infarction due to coronary thrombosis in the short term. Therefore, the clinical manifestations of this type are quite different, and the age of onset is relatively young, which is more common in men.

III. Deteriorating angina pectoris: Patients with stable angina pectoris often experience changes in frequency, severity, time limit, and inducing factors within 3 months, showing progressive deterioration. This type of angina pectoris activity tolerance is significantly reduced, can even occur in the resting state, the pain time is prolonged, and the sensitivity to nitroglycerin is reduced. It may be due to increased lipid infiltration in atherosclerotic plaque or plaque rupture, local platelet aggregation, and it may develop into acute myocardial infarction.

Fourth, supine angina pectoris: It is characterized by occurring often at rest or when asleep, because it often occurs at night, sitting or standing during an attack can reduce symptoms. Patients with this type of angina often have a long history of coronary heart disease, due to long-term myocardial ischemia, a certain degree of myocardial sclerosis and cardiac insufficiency; or due to increased venous return during supine, cardiac workload and cardiac oxygen consumption increase related. Can progress to myocardial infarction or sudden death.

V. Variant angina pectoris: The onset is similar to supine angina pectoris, but the ST segment of the corresponding lead of the electrocardiogram is elevated during the onset, and the ST segment of the corresponding lead is depressed, which is induced by coronary spasm. .

6. Mixed angina pectoris: Patients can have angina pectoris when myocardial oxygen demand increases, or angina pectoris when there is no significant increase in myocardial oxygen demand. Coronary artery stenosis reduces the amount of coronary blood flow reserve, and this decrease in blood flow reserve is not fixed, often caused by further fluctuations.

1. If a patient with coronary heart disease suddenly develops pain in the precardiac area, chest tightness, shortness of breath, and angina pectoris at home, he should lie down immediately with nitroglycerin tablets under his tongue. If one tablet does not solve the problem, take another tablet. If the seizure has been relieved, you need to lie down for an hour before getting out of bed.

2. If the patient is in a dire condition and has chest pains that are pale and sweaty, this may not be an ordinary angina pectoris attack, but I am afraid that a myocardial infarction has occurred. At this point, wrap the isoamyl nitrite with a handkerchief, break it, move it about 2.5 cm near the nose, and inhale the gas. If the patient is nervous, give one tablet orally. On the other hand, you should contact the emergency center immediately. You must not move the patient at will. If you are near the hospital, you can use a stretcher or a bed to lift it away.

3. If the patient has tachycardia during angina pectoris, add 1-2 lactic acid coredine tablets based on nitroglycerin.

First Aid Precautions

When the coronary heart disease angina pectoris or myocardial infarction, the patient must be supine, do not move at will, do not rush to the doctor, not to help the patient to the hospital. Can be rescued at home according to the above method. If it is an angina pectoris, it can be relieved after treatment. If myocardial infarction is not resolved, you must contact the emergency center.

What Are the Most Common Causes of Mild Chest Pain?

Chest pain after exhaustion

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