What Is the Treatment for Spinal Cord Compression?

The etiology of the lesion can be divided into three categories:

Spinal cord compression

Spinal cord compression (compressivemyelopathy) is a group of intraspinal diseases with a space effect. Changes in the spinal cord after compression are related to the location of the compression, the nature of the external compression, and the rate of occurrence. With the development and expansion of the etiology, the spinal cord, spinal nerve roots and their supply vessels are under pressure and become more and more severe. Once the compensatory capacity is exceeded, pathological changes such as spinal cord edema, degeneration, and necrosis will eventually be caused. Tube obstruction causes limb movement, sensation, reflexes, sphincter function, and skin nutritional dysfunction below the compression plane, which seriously affects the patient's life and work ability.

Affected area

whole body

Related diseases

Omentitis, spinal cord compression, multiple myeloma, ankylosing spondylitis, spinal tuberculosis, spina bifida fracture, scoliosis, spinal cord injury, spinal cord tumor, idiopathic scoliosis, congenital scoliosis, scleritis, abscess, neurofibromas, ankylosing spondylitis and Its associated uveitis meningioma spinal vascular malformation spina bifida and related deformities cryptic spina bifida multiple myeloma disease nephropathy intraspinal hemorrhage intraspinal abscess multiple elderly myeloma ankylosing spondylitis scleritis intramedullary tumors in children Spina bifida

Related symptoms

Incontinence inflammation Inflammation Sensitivity Sensory disorders Bone metastasis Muscle spinal cord Horsetail involved Spinal cord Thalamic tract Compression Spinal cord Compression Spinal cord compression Tendon reflex Cough Urinary skin Dryness Edema Paralysis Inability to shock Autonomic dysfunction

Affiliated Department

Department of Internal Medicine

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The etiology of the lesion can be divided into three categories:

First, spinal diseases: can be caused by vertebral fractures, dislocations, disc herniation, spinal stenosis, spinal tuberculosis, spinal primary tumor function metastasis and so on.

2. Spinal and extraspinal lesions: extramedullary tumors such as neurofibromas and meningiomas, spinal arachnoiditis, spinal vascular malformations, and epidural abscesses.

Third, spinal cord lesions: such as tumors, tuberculomas, and bleeding.

In general, the development of clinical symptoms is:

I. Symptoms of spinal nerve root compression: Burning, tearing or drilling pain often occurs due to the compression of one or more posterior roots of the spinal nerve, and it can be radiated to the corresponding skin segment. It can be caused when the spine is active, coughing or sneezing. Causes the pain to aggravate, and appropriate changes in position can be reduced. This first symptom of root pain often has important diagnostic significance. Spinal cord collapse caused by dural meningitis, extramedullary tumors, especially neurofibromas, and various primordial calendars, and root pain is often more prominent. Allergic or abnormal areas can often be found in the area of root pain. If the function is impaired, it can cause segmental dullness. If the lesion is located on the ventral side of the spinal cord, it can stimulate and damage the anterior root of the spinal nerve, causing segmental muscle spasm and muscle atrophy.

Second, the symptoms of spinal cord compression:

(A) Movement disorders. When the anterior horn of the spinal cord is compressed, symptoms of segmental submotor neuron palsy can occur, which are manifested by atrophy, weakness, and muscle fibrillation of limb or trunk muscles within the area dominated by the damaged anterior horn. When the corticospinal tract is damaged, it causes spastic paralysis of the limbs below the compression plane-paralyzed limbs have increased muscle tone, hypertenoid reflexes, and positive pathological reflexes.

(B) Sensory disorders. The plane of sensory disturbance is often of great reference value for localization of lesions.

(3) Abnormal reflection.

(D) autonomic dysfunction: dry skin below the lesion level, less sweat, rough toenails, and edema of the limbs. Chronic compression lesions above the lumbosacral spinal cord, early urgency is difficult to control; if it is a severely impaired shock period, automatic urination and defecation are lost, and later transition to incontinence. The lumbosacral spinal cord lesions are characterized by urine and stool retention.

3. Spine Symptoms: There may be signs of tenderness, tenderness, deformity, restricted movement, etc. at the site of the lesion.

4. Spinal canal obstruction: Compression myelopathy can cause incomplete or complete obstruction of the subarachnoid space of the spinal cord.

Spinal cord compression: Spinal cord compression occurs after hepatocellular carcinoma bone metastases. Fracture displacement, crushed bone fragments and broken intervertebral discs squeezed into the spinal canal can directly compress the spinal cord, and the folded yellow ligament and rapidly formed hematoma can also compress the spinal cord, causing the spinal cord to be compressed. Spinal cord compression occurs after hepatocellular carcinoma bone metastases. Fracture displacement, crushed bone fragments and broken intervertebral discs squeezed into the spinal canal can directly compress the spinal cord, and the folded yellow ligament and rapidly formed hematoma can also compress the spinal cord, causing the spinal cord to be compressed.

Compression of the thalamus of the spinal cord: When the spinal cord is compressed, dyskinesia precedes sensory disturbance. In addition to compression of the spinal cord tissue, it may also be accompanied by blood circulation disorders, cerebrospinal fluid dynamics disorders, and concurrent inflammation and adhesion. As a result, clinical manifestations appear diverse and complex. The spinal cord and spinal nerve roots in the plane of the primary spinal canal are compressed.

Spinal and horsetail involvement: Involvement of the spinal cord, cauda equina, or nerve roots is one of the symptoms of spinal cord injury. There are more and more patients with spinal injuries, depending on the mechanism of damage, and the classification is different, so it is also difficult to diagnose. However, in fact, as long as the local pathological anatomy can be grasped, and a comprehensive collection of trauma history, symptoms and signs can be seen and comprehensively analyzed and judged, it is not difficult to obtain a correct diagnosis in most cases. On this basis, treatment problems are also easy to solve. For certain clinical diagnosis, CT, MRI, CT plus myelography, CTM and other imaging examination methods can be used.

I. Etiology treatment: Surgery or (and) medication for the cause.

Second, symptomatic treatment: keep the skin dry, avoid bedsores, keep the urination unobstructed, prevent urinary tract infections, massage and exercise the paralyzed limbs; for high paralysis, pay attention to respiratory protection and prevent lung infections.

Third, insist on regular medical examinations on time every year.