What Are Common Causes of Shortness Of Breath and Fatigue?

Respiratory muscle fatigue (also known as respiratory muscle weakness) refers to respiratory muscle contraction caused by multiple reasons that cannot produce the thoracic pressure required to maintain a certain alveolar ventilation. The muscle itself can show muscle strength and / or contraction rate Decline, and this decline in ability can be restored by rest. Failure to recover after a break is called weaknees. Diaphragm is the main respiratory muscle. Therefore, respiratory muscle fatigue in the narrow sense actually refers to diaphragm fatigue.

Respiratory muscle fatigue

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Respiratory muscle fatigue (also known as respiratory muscle weakness) refers to respiratory muscle contraction caused by multiple reasons that cannot produce the thoracic pressure required to maintain a certain alveolar ventilation. The muscle itself can show muscle strength and / or contraction rate Decline, and this decline in ability can be restored by rest. Failure to recover after a break is called weaknees. Diaphragm is the main respiratory muscle. Therefore, respiratory muscle fatigue in the narrow sense actually refers to diaphragm fatigue.

Respiratory muscle fatigue in most patients is caused by the disease itself, and sepsis has been found to cause multiple organ failure,

Respiratory muscle fatigue or respiratory muscle dysfunction is common in patients with chronic lung disease and ICU admissions. For a long time, the problem of respiratory muscle fatigue or failure has not been taken seriously. The timely detection and treatment of respiratory muscle fatigue can correct respiratory mechanics abnormalities, reduce respiratory work, improve oxygenation, and shorten the time of mechanical ventilation. Therefore, in recent years, the evaluation of respiratory muscle function in critically ill patients has become one of the important contents of intensive care.

Laboratory inspection

1.Maximum suction

1. According to the medical history, there is a history of basic respiratory diseases.

2. Clinical manifestations include accelerated breathing rate and out of breath.

3. Increased creatine phospholipid kinase in blood, which can be diagnosed according to 3 items.

The prolongation of muscle relaxation is more common in patients who have been using muscle relaxants for a long time or in large quantities, or at the same time, drugs that affect neuromuscular function. The incidence in ICU is about 5%, which can prolong ICU hospitalization time. The incidence of NBA-induced acute myopathy is lower than that of prolonged muscle relaxation. It is most common when acute doses of steroids and NBA are used simultaneously in the acute episode of asthma. In a prospective clinical study, Leatherman observed 25 asthmatic patients receiving both Vecuronium and corticosteroids and found that 19 patients had creatine phosphate

Prevention: If the respiratory muscles are fatigued or functionally depleted, the fire canister net plays an important role in the occurrence of respiratory failure. First, the causes of respiratory muscle dysfunction should be corrected and removed. The general principles are: ensure that the respiratory muscles have sufficient energy supply, including supplementary nutrition correct

Fatigue breathing muscles can restore function after rest. At present, positive pressure ventilation is usually used to replace or partially replace the respiratory muscles to complete the ventilation, so that the tired respiratory muscles can rest. Oral and nasal masks can be used for non-invasive positive pressure ventilation. For patients with unconsciousness, uncooperative respiratory secretions, and multiple hemodynamic instability, tracheal intubation should be used to establish artificial airway ventilation. At present, patients with chronic respiratory failure are breathing. Muscle dysfunction mostly advocates non-invasive positive pressure ventilation. It has also achieved good results in patients with chronic respiratory failure such as thoracic deformities of chronic neuromuscular diseases. However, the effects are different in patients with COPD. The main question is whether non-invasive ventilation is really reduced. The activity of the diaphragm muscles allows the diaphragm muscles to fully rest.The length of ventilation time, the size of the assisted ventilation pressure, the severity of the patient's underlying disease, and the medication can all affect the judgment of the efficacy of noninvasive ventilation. Most opinions tend to be applied correctly. Good non-invasive ventilation can help many patients avoid tracheal intubation by reducing the work of breathing muscles. Of course, there are other mechanisms for non-invasive ventilation to improve the condition, such as: readjusting the sensitivity of the respiratory center to carbon dioxide, reducing the effect of hypoxia and CO2 retention on respiratory muscle function by improving blood gas, and excessive rest can cause respiratory muscle atrophy and cause breathing. Machine dependence. It is difficult to determine the ideal limit for complete rest and load of respiratory muscles in clinical practice. The general principle is to control ventilation or high-level pressure support ventilation for 24-48 hours. After the fatigued respiratory muscles are fully rested, the intensity of ventilation support should be reduced in time Gradually increase the patient's respiratory load and actively prepare for weaning. ^[1]