What Are the Common Causes of Green Pus?

Pseudomonas aeruginosa, also known as Pseudomonas aeruginosa (scientific name: Pseudomonas aeruginosa), was first isolated from wound pus by Gersard in 1882. Only one-way movement. It is an opportunistic bacterial infection, and it is also an opportunistic infection to plants. After infection, it is named green because of disease materials such as pus and exudate.

The scientific name of Pseudomonas aeruginosa is Pseudomonas aeruginosa, of which Pseudomo

It is about 1.5 3.0 × 0.5 0.8umg in width. Single, paired or occasionally short chains, filamentous morphology can be seen in broth cultures. The mycelium has 1-3 flagella and is active. No spores, can form

Very strong ability to break down proteins, but low ability to ferment sugars, break down glucose, primary gum, mono-toffee, mannose

Pseudomonas aeruginosa has bacterial cell o antigen and flagella h antigen. One is the o antigen

The bacteria is more resistant to chemicals than normal

Pseudomonas aeruginosa is an immunocompromised opportunistic pathogen that generally affects the lungs and urinary tract, or causes burns, wounds, and other blood infections, such as sepsis. Although rare, Pseudomonas aeruginosa can also cause pneumonia. There is also a chance to cause corneal infection of the eye if the contact lens is not completely cleaned. Many studies on respirator-associated pneumonia have pointed out that Pseudomonas aeruginosa is one of the bacteria that needs to be isolated. Pyocyanin-a blue-green toxic metabolite, is one of the pathogenic factors of Pseudomonas aeruginosa, and can cause C. elegans to die under oxidative stress, but some studies have suggested that salicylic acid Inhibits the production of chloropyridin. 10% of hospital infections are caused by Pseudomonas aeruginosa. The lungs of patients with cystic fibrosis are the first to be infected with Pseudomonas aeruginosa. It is also one of the bacteria that cause dermatitis in the absence of proper treatment of water quality. It is also the most common bacterium that causes burn infections.

Pseudomonas aeruginosa can produce a variety of pathogenic substances, mainly

Pseudomonas aeruginosa is inherently resistant to most antibiotics, and can rapidly generate resistance mutations. Generally, two antibiotics (aminoglycoside antibiotics, -lactam antibiotics or quinolone antibiotics) are used in combination with aggressive antibacterial treatment. General treatment must be assisted by laboratory sensitivity rather than empirical selection of antibiotics. If antibiotics have been used, their reproduction must be tried to understand and review the effectiveness of antibiotics frequently.

Antibiotics that are effective against Pseudomonas aeruginosa include:

Aminoglycoside antibiotics (gentamicin, amikacin, tobramycin)

Quinolones (ciprofloxacin and colabitux, but excluding moxifloxacin)

Cephalosporins (ceftazidime, cefepime, cefpirome, but not cefuroxime, ceftriaxone, cefotaxime)

Piperacillin, Ticarcillin (Pseudomonas aeruginosa is essentially resistant to all other penicillins)

Carbapenems (donipenem, meropenem, yampenem, but not ertapenem)

Aztreonam

With the exception of fluoroquinolone, all of the above antibiotics must be administered by injection. Therefore, the use of fluoroquinolone is strictly prohibited in some hospitals to prevent P. aeruginosa from developing corresponding resistance. ^[2]