What Is the Connection Between Adiponectin and Diabetes?

Adipose tissue is mainly composed of a large number of agglomerated adipocytes. Adiponectin / ADPN is an endogenous biologically active polypeptide or protein secreted by adipocytes. Adiponectin is an insulin-sensitizing hormone that can improve insulin resistance and arteriosclerosis in mice; studies in humans have found that adiponectin levels can predict type II diabetes And the development of coronary heart disease, and has shown anti-diabetic, anti-atherogenic and inflammatory potential in clinical trials.

Researchers have discovered a new compound that regulates adiponectin
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Adiponectin is not only made by
Kondo et al. Discovered by extensive genome scan
In October 2009, Elsevier reported that the first randomized double-blind placebo-controlled human trial in humans has shown that astaxanthin can consume human triglycerides and increase high-density lipoprotein HDL and adiponectin. Research Units: Pharmaceutical Laboratory, Kashiwa Hospital, Tzuhui University, Chiba, Japan; International Institute of Nutrition and Metabolism, Tzuhui University, Tokyo, Japan.
BACKGROUND: Astaxanthin has been reported to significantly improve dyslipidemia and metabolic disorders in animals, but the effect of astaxanthin on human lipids is not well known.
Methods: The natural astaxanthin dosage was 0, 6, 12, 18 mg / day for 12 weeks, and a placebo control was used. Samples were selected.
Astaxanthin increases adiponectin
Random selection: 61 non-obese people aged 25-60 years, without diabetes or hypertension, fasting serum triglycerides 120-200mg / dl.
Results: Before and after the control test, body mass index (BMI) and LDL low-density lipoprotein had no effect at the above doses. However, triglycerides were reduced by 30-50%, and high-density ester protein (HDL) was significantly increased by 20-30%. Multi-sample comparisons showed that triglycerides were significantly reduced by 30-50% in the 12 and 18 mg / day groups, and high density ester protein (HDL) was significantly increased by 20-30% in the 6-12 mg / day dose group. Serum in the 12-18 mg / day dose group Adiponectin increased, and changes in adiponectin were positively correlated with changes in HDL, and were not related to indexes such as age and fitness.
The upper graph shows that adiponectin increases the most when astaxanthin reaches 12 mg / day. The lower graph shows that HDL also increased significantly with the increase of adiponectin.

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