What Is the Tricuspid Valve?
Three triangular valves are attached to the fibrous stent ring composed of dense connective tissue at the right atrioventricular opening, which is called the tricuspid valve or right atrioventricular valve. The tricuspid valve acts like a "one-way valve", ensuring that blood circulation flows from the right atrium to the right ventricle and through a certain flow.
Tricuspid valve
Three triangular valves are attached to the fibrous stent ring composed of dense connective tissue at the right atrioventricular opening, which is called the tricuspid valve or right atrioventricular valve. The tricuspid valve acts like a "one-way valve" to ensure blood circulation by
The right atrium wall of the heart is thin. According to the direction of blood flow, the right atrium has three entrances and one exit. The entrance is up
The tricuspid valve acts like a "one-way valve", ensuring that blood circulation flows from the right atrium to the right ventricle and through a certain flow. When the right ventricle contracts, the heart squeezes the blood in the room and the blood hits the valve. The tricuspid valve is closed and blood is not poured into the right atrium. Right ventricle
Tricuspid atresia
Tricuspid atresia Tricuspid atresia is a type of cyanotic congenital heart disease. Tricuspid atresia
1 to 5%. Ranked third after cyanotic congenital heart disease relay Fallot tetralogy and aortic dislocation. The main pathological changes are tricuspid valve atresia or absence of tricuspid valve, oblong foramen ovale or atrial septal defect.
Tricuspid insufficiency
Tricuspidinsufficiency is rare in the leaflet itself, and is mostly caused by pulmonary hypertension and Tricuspid insufficiency
Caused by tricuspid valve dilatation. Due to congenital or acquired factors causing tricuspid valve disease or tricuspid annulus dilation, the tricuspid valve is not fully closed when the systole is not fully closed. The disease is both functional and organic. The former is mostly secondary to lesions that cause right ventricular dilatation, and the incidence is quite high, such as primary pulmonary hypertension, mitral valve disease, pulmonary valve or funnel stenosis, and right ventricle. Myocardial infarction and so on. The latter can be congenital anomalies such as Ebstein's malformation and common atrioventricular channels, or acquired lesions such as rheumatic inflammation, coronary artery disease caused by tricuspid papillary muscle dysfunction, trauma and infective endocarditis. The prognosis of the disease depends on the nature of the underlying cause and the severity of heart failure. The prognosis of patients with primary pulmonary hypertension and chronic pulmonary heart disease is often worse than those caused by mitral valve disease or atrial septal defect. . Medical treatment can alleviate the symptoms, and surgery can cure it.
Tricuspid valve malformation
Tricuspid valve malformation is a rare congenital heart malformation. First reported by Ebstein in 1866 Tricuspid valve malformation
For example, it is also called Ebstein deformity. The incidence of congenital heart disease accounts for 0.5 to 1%. Tricuspid valve malformation refers to the tricuspid valve malformation. Its posterior and septal valve positions are lower than normal. It is not at the level of the atrioventricular ring and moves down to the right ventricle wall near the apex. Normally large, while the right ventricle is smaller than normal, with tricuspid regurgitation. Such malformations are often associated with open oval holes or atrial septal defects and pulmonary artery stenosis. Due to the large amount of blood in the right atrium and increased pressure, part of the blood contained in the right atrium flows into the left atrium through the atrial septal defect or oval hole, and part of it still enters the right ventricle through the tricuspid valve. Due to pulmonary artery stenosis, the blood volume entering the pulmonary circulation is reduced. Therefore, the amount of arterial blood returned to the left atrium is also small. At this time, it is mixed with the venous blood shunted from the right atrium and enters the left ventricle and systemic circulation through the mitral valve. See tricuspid valve malformation for details.
