What Are the Most Common Causes of Heartburn and Dizziness?

Neurogenic Gastritis is a type of gastritis caused by persistent nerve tension.

Chinese name: Neurogastritis

Foreign name: Neurogenic Gastritis

Overview of neurogastritis

Neurogastritis is a type of gastritis, a stomach disease caused by a patient's long-term high-intensity mental stress and tension. Neurogastritis is generally not a very serious disease.

Causes of neurogastritis

The stomach responds too strongly to the stimulus. After the cause is eliminated, it will heal on its own, but persistent disease can lead to chronic gastritis.

Neurogastritis can usually be treated by taking antacids, sedatives and other drugs. The disease is usually caused by a high level of mental stress. Patients should be able to learn to relieve stress and relax in the ordinary life to face life.

Neurological gastritis

The more common symptoms of neurogastritis are loss of appetite, burning stomach, stomach pain, acid reflux, etc., and severe symptoms such as dizziness and vomiting. Patients should go to the hospital in time when pre-symptoms appear.

1. No appetite, stomach burning, stomach pain, accompanied by vomiting;

2. Similar to the symptoms of acute and chronic gastritis, vomiting, pain, and vomiting blood in severe cases.

Neurogastritis complications

(A) gastric ulcer:

Gastric ulcer refers to a localized defect in the stomach. The cause may be due to delayed gastric emptying, retention of the gastric antrum, release of gastrin to cause an increase in gastric acid, and the formation of ulcers. It manifested as middle and upper abdominal pain without clear rhythmicity and periodicity. Antacids have poor efficacy. The base acid is not high or low, and the cancer rate is 5%. 90% of ulcers are on the lesser side of the stomach. Indications for surgery: 3 months of non-healing in medical treatment; recurrent episodes; large ulcers with a diameter of more than 2 cm; canceration is not excluded; Advocate complete type I gastric resection. Those with good curative effect were above 90%.

Gastric acid-secreting gastric parietal cells are dominated by the vagus nerve, so vagotomy, that is, cutting the vagus nerve trunk at the esophageal hiatus to reduce gastric acid secretion, is often used to treat certain peptic ulcers (gastric mucosal damage is usually related to Helicobacter pylori infection) . Studies show that 9/10 of gastric ulcers are caused by H. pylori infection and can be treated with antibiotics. Under normal circumstances, mucus covers the surface of the mucosa, forming a barrier between the mucosal cells and gastric acid. If this barrier is insufficient, gastric acid can erode the mucous membranes and cause ulcers. If the ulcers erode into the arteries, they can cause life-threatening bleeding. Vaginectomy can be performed with the removal of the ulcer (sinectomy). During selective vagalectomy, the anterior and posterior branches of the vagus nerve that dominate the stomach are severed, but the branches of the vagus nerve that dominate the pylorus, biliary tract, bowel, and celiac plexus are retained. Parietal cell vagotomy attempts to more accurately cut off the branches of the vagus nerve distributed in the area where parietal cells are located, affecting only gastric acid-secreting cells, while other structures in the abdomen still retain vagal innervation.

A posterior gastric ulcer may penetrate the stomach wall and affect the pancreas, leading to involved pain in the back. At this time, ulcers involving the spleen artery can cause severe bleeding and remain in the peritoneal cavity. Pain from the stomach stimulates the afferent fibers through the viscera to walk with the sympathetic nerves. After bilateral vagus nerve amputation, the persistent pain after peptic ulcer recurrence is still obvious; while patients undergoing bilateral sympathectomy may have perforated ulcers without pain.

(B) Duodenal ulcer:

Occurs in round or oval tissue defects of the duodenum. It occurs most often in the ampulla. Its occurrence and development are mainly related to the digestion of gastric juice. It is clinically characterized by recurrent episodes of abdomen and rhythmic pain. The typical pain is mostly fasting pain and nocturnal pain. The pain occurs 2 to 3 hours after eating. The pain is relieved after continuous eating or taking an antacid, that is, the pain-eating-relief rule is formed. When penetrating ulcers develop, they show persistent severe pain. The main complications are massive bleeding, perforation, and obstruction. Diagnosis is based on X-ray barium meal examination and duodenoscopy. Treatment mainly neutralizes gastric acid and inhibits gastric acid secretion. This disease can be seen in older children.