What Is Mild Dyslexia?

Mild mental retardation is a synthesis of mental development that is caused by abnormalities in the patient's genetic (gene) system caused by environmental or genetic influences. It is a synthesis that stops development at various stages of life before birth, during birth, or in early life due to various reasons. symptom. Mild mentally handicapped people have extremely poor learning abilities and have reduced ability to adapt to life. They are mainly manifested in their inability to understand the problem correctly, not to think flexibly, and the slow speed of solving the problem. The disease has a large number of patients worldwide.

Mildly mentally retarded

Mild mental retardation is a synthesis of mental development that is caused by abnormalities in the patient's genetic (gene) system caused by environmental or genetic influences. It is a synthesis that stops development at various stages of life before birth, during birth, or in early life due to various reasons. symptom. Mild mentally handicapped people have extremely poor learning abilities and have reduced ability to adapt to life. They are mainly manifested in their inability to understand the problem correctly, not to think flexibly, and the slow speed of solving the problem. The disease has a large number of patients worldwide.

Chinese name

Mildly mentally retarded

which performed

Cannot understand the problem correctly

Alias

Mild mental retardation

Features

Slow

Mild mental retardation (alias: "mild mental retardation" or "mild brain hypoplasia") refers to intelligence whose intelligence development is slightly behind or below the normal level of the same age. Main manifestations of patients with the disease

(1) Mild mental retardation

Mildly mentally retarded children are more developed in all aspects

Mild intellectual impairment, IQ of 50 to 70, belongs to the stupid type.

Infancy is the fastest period of brain, mental and physical development. From the beginning of the newborn, to the age of 7, the brain weight is close to the adult brain weight.

Some adults with mildly mentally handicapped people may experience some increase in intelligence after they reach adulthood. The reason may be that experience and experience may increase with the passage of time; in addition,

There are also some mentally handicapped people with special skills, such as

(1) Patience teach him or her literacy, repeat the basic method of Pinyin repeatedly, and make illustrations.

(2) Take him or her out for a field outing, get to know nature, and make his soul suffer

Chinese medicines with higher safety factor for nootropic drugs and health products:

-aminobutyric acid tablets,

Optional for children with mental illness:

Electroacupuncture and Acupuncture

Simultaneously pierce with a needle

(1) any drug is there

(1) There are several methods to assess the intelligence level of patients:

: Stanford-Binet Intelligence Scale

IQ (IQ) = Mental age (that is, the mentality of cognition at actual age) ÷ Actual age × 100

:

(1) Failure of Intelligence Test

(2) Lack of emotional performance

(3) Grow up in an abnormal environment (excluding some patients with moderate or higher psychological damage)

(4)

Drugs that raise choline levels in the brain

Example: Choline

Neuropeptide plays a very important role in central nervous system information transmission, it has

Mathematics is a discipline with strong logical thinking, and the process of learning mathematics is a

Mentally retarded children not only lag behind their normal children's development level, but also because of their overall psychological development

(1) Not treated during development or pre-development.

(2) Possible interaction of drugs.

(3) Blind treatment of unknown etiology.

(1) Good interpersonal relationship (except Stockholm syndrome, except for special circumstances, special cultural style).

(2) Self-learning ability can reach ordinary high school level 100 points paper, the score is not less than 80 points (under the guidance of a book or teacher)

(3) Have a certain psychological defense ability.

Generally speaking, mildly retarded children

(1) Neural Stem / Progenitor Cells: Transplantation of neural stem / progenitor cells into the brain can survive the damaged hippocampus and facilitate the formation of synapses. Therefore, it can be proven that exogenous cells can form effective neural networks Functional connection.

(2)

(1) In general, the left side of the brain of patients with mild dyslexia is larger than the right side. The autopsy found mild

Mild mental retardation and dementia (Alzheimer's disease)

Mild mental retardation occurs before the age of 18, and dementia occurs after the age of 18 (but the medication has many similarities, basically the same; except for some special pathologies).

Mild mental retardation and learning disability syndrome

Children with learning disability syndrome also have learning difficulties and are easily mistaken for intellectual problems. However, some of them are normal, but others are abnormal. Some can be juxtaposed with mild mental retardation, which is difficult to identify. But the most important point of learning disability syndrome is

(1) The DS cell adhesion molecule (DSCAM) gene is located at 21q22.2-22.2, with a length of 75Mb, and encodes a cell adhesion molecule. The gene is expressed to varying degrees in adult brain tissue. Tissue in situ hybridization of the mouse DSCAM gene shows that the gene is expressed in the central and peripheral nerves, suggesting that DSCAM is involved in neural system differentiation and is related to DS central and peripheral nerve defects. . DT-PCR showed that the gene was also expressed in human embryos and fetal heart tissues at 7.5 to 10 weeks, and its overexpression was also related to the occurrence of congenital heart disease in DS.

(2) Activity-dependent neuroprotective protein (ADNP) genes are mostly expressed in the hippocampus, cerebral cortex and cerebellum. ADNP is composed of 828 amino acids with a PI of 5.99 and is widely distributed in all organs. Many studies have shown that ADNP is a new type of heat shock protein.

(3) The DSCRI gene is located at 21q21.1 to 22.2 and is highly expressed in fetal and adult hearts. A series of experiments have shown that DSCRI is involved in regulating Calcineurin activity in vivo, and Calcineurin can regulate many physiological processes, such as neurotransmitter and hormone release, synapse formation and gene transcription, so it is speculated that DSCRI may be related to DS learning and behavior changes .