What Is the Superior Oblique?
It is the striated muscle that starts from the upper and inner sides of the total tendon ring and is attached to the midline of the eyeball or later on the lateral sclera of the equator. Its muscle length is about 60 millimeters, which is the longest muscle in the extraocular muscles. It advances along the upper side of the orbit to the frontal block of the inner edge of the orbit. The tendon becomes round. After passing through the block, it folds to the outside of the eyeball. And at an angle of 51 ° to the visual axis. The tendon flattened below the superior rectus muscle, and after crossing the equator of the eyeball, it stopped on the lateral sclera. The front end of the attachment line is about 5 mm behind the lateral end of the superior rectus muscle attachment line; 13 mm from the limbus, and the back end of the attachment line is 6.5 mm from the optic nerve; the attachment line is curved, the muscle width is 10.7 mm, and the tendon is 30 mm long. 9.4 mm wide; innervated by the tackle, 26 mm away from the tackle as the innervation point. Function: Due to the segment behind the muscle block, the direction of travel is at an angle of 51 ° to the visual axis. In the first eye position, the main role is internal rotation, and the secondary role is to turn down and out; When the position is 51 °, the direction of muscle traction is consistent with the visual axis, and only has a downward turning effect; when the outward rotation of the eyeball is 39 ° from the first eye position, the direction of the muscle traction is at right angles to the visual axis, and only the internal rotation is exerted. Can be accompanied by external movement.
- Chinese name
- Superior oblique
- Foreign name
- obliquus superior
- From
- Total tendon ring around the optic canal
- full length
- 62mm
- It is the striated muscle that starts from the upper and inner sides of the total tendon ring and is attached to the midline of the eyeball or later on the lateral sclera of the equator. Its muscle length is about 60 millimeters, which is the longest muscle in the extraocular muscles. It advances along the upper side of the orbit to the frontal block of the inner edge of the orbit. The tendon becomes round. After passing through the block, it folds to the outside of the eyeball. And at an angle of 51 ° to the visual axis. The tendon flattened below the superior rectus muscle, and after crossing the equator of the eyeball, it stopped on the lateral sclera. The front end of the attachment line is about 5 mm behind the lateral end of the superior rectus muscle attachment line; 13 mm from the limbus, and the back end of the attachment line is 6.5 mm from the optic nerve; the attachment line is curved, the muscle width is 10.7 mm, and the tendon is 30 mm long. 9.4 mm wide; innervated by the tackle, 26 mm away from the tackle as the innervation point. Function: Due to the segment behind the muscle block, the direction of travel is at an angle of 51 ° to the visual axis. In the first eye position, the main role is internal rotation, and the secondary role is to turn down and out; When the position is 51 °, the direction of muscle traction is consistent with the visual axis, and only has a downward turning effect; when the outward rotation of the eyeball is 39 ° from the first eye position, the direction of the muscle traction is at right angles to the visual axis, and only the internal rotation is exerted. Can be accompanied by external movement.
Superior oblique tendon sheath syndrome
- 1. Essence is a symptom of unknown cause, which may be congenital anomalies or defects, with eye movement disorder as the main symptom. Also known as Brown superior oblique tendon sheath syndrome; Brown syndrome; tendon sheath adhesion syndrome (tendon sheathadherence syndrome). Onset in childhood, which gradually increases with age. It manifests as head tilted back, drooping eyelids on both sides, and narrowed eyelids. Eye movement is limited, and it is almost fixed in a position to look down. May be accompanied by reduced elasticity of the binding membrane, choroidal defects and so on. Because it is a congenital anatomic defect, no satisfactory treatment is available. The eigen does not affect life, nor does it affect life.
- 2. Eye movement limitation disease caused by overtightening of the superior oblique tendon sheath. The patient's eyes could not be turned up when turned in, and normal or almost normal when turned up and out. The palpebral fissure occasionally enlarges during introversion, and the eyeball is turned downward due to the mechanical pull of the superior oblique muscle. The ipsilateral superior oblique was not hyperactive. Exotropia is often noticeable when looking up. Most patients were in the right position when they were in place, but the strabismus occurred with the course of the disease, and the face turned to the compensatory head position of the contralateral eye. If faced upright when in place, no abnormal head position, no treatment is needed. If the affected eye is significantly lowered or the head is abnormal, surgery to remove the abnormal upper oblique tendon sheath is required.