What Is the Connection Between Severe Headaches and Nausea?

(I) Causes of Onset

Headache and vomiting

Headache accompanied by severe vomiting is suggestive of increased intracranial pressure. Migraine can be seen as a relief of headache after vomiting. Generally, increased intracranial pressure is one of the common factors that stimulate the vomiting center. Another manifestation of increased intracranial pressure is headache. Headache with nausea, vomiting, diarrhea, and fever can be seen in acute gastroenteritis. Headache with severe jet-like vomiting is common in patients with elevated intracranial pressure. Headache with vomiting, but the vomiting is not severe and the headache is significantly relieved after vomiting is a characteristic of migraine.

Affected area

head

Related diseases

Abdominal Migraine Syndrome Amenorrhoea Irregular Menstruation Thrombosis Benign Intracranial Hypertension Postpartum Three Acute Intercourse Headache Cerebral Esophagus Deficiency Deficiency Cold Vomiting Blood Deficiency Headache Swelling

Related symptoms

Localized signs venous embolism intracranial pressure increased meningitis granulomatous edema headache

Affiliated Department

Surgical neurosurgery

Related inspections

Hepatitis A Antigen Hepatitis A Virus Antigen (HAVAg) Cerebrospinal fluid Calcium Cerebrospinal fluid Tuberculous meningitis antibodies Cerebrospinal fluid Cysticercosis Indirect hemagglutination test Cerebrospinal meningococcal antigen Cerebrospinal fluid Lead Cerebrospinal fluid Tryptophan test Urine p-hydroxyphenylpyruvate Dopamine urine magnesium Urine manganese arginyl succinate lyase cerebrospinal fluid lysozyme bacteriological examination Bacteriological examination of blood and bone marrow specimens

(I) Causes of Onset

The cause can be endocrine and metabolic disorders, intracranial venous sinus thrombosis, drugs and toxins, as well as primary benign intracranial hypertension, that is, those with unknown reasons.

(Two) pathogenesis

The pathogenesis of this disease is unknown, and it is intracranial hypertension that develops within weeks or months. As for the direct cause of the increased intracranial pressure is due to the swelling of the brain parenchyma or the cerebrospinal fluid changes, it is still inconclusive. Most people think that it is caused by cerebrospinal fluid absorption disorders, but there is not much evidence.

Karahalios et al. (1996) found that all patients with benign intracranial hypertension had an increase in cerebral venous pressure. In this case, cerebrospinal fluid absorption was blocked, which could lead to an increase in intracranial pressure. However, an increase in intracranial pressure was caused by an increase in intracranial pressure. The cause or result is unclear. During continuous cerebrospinal fluid monitoring in patients with benign intracranial hypertension, it was found that there was an uninterrupted and irregular fluctuation in cerebrospinal fluid pressure. When the pressure increased to a plateau for 20 to 30 minutes, it suddenly dropped to normal levels. It is like draining the increased CSF (Johnston and Paterson, 1974). A considerable number of patients have reported irregular menstruation or amenorrhea, some are pregnant women, some have endocrine system dysfunction, others have taken tetracycline, indomethacin, oral contraceptives or other hormones, and have been reported to be associated with vitamin A poisoning. . It is inferred that the above conditions are related to benign intracranial pressure, but there is no substantial evidence.

The diagnostic criteria are as follows:

1. There are symptoms and signs of increased intracranial pressure.

2. Nerve examination showed no localized signs.

3. Except for the increase of cerebrospinal fluid pressure, there is no abnormality in neuro-diagnostic examination (no deformation, displacement or obstruction of ventricle system).

4. The patient is conscious.

5. There are no other causes that can cause increased intracranial pressure.

6. If the cerebrospinal fluid examination is abnormal, the diagnosis is unsuccessful.

There are mainly dural venous sinus embolism, diffuse glioma disease, cancerous meningitis, granulomatous meningitis, and micro cerebral arterial malformations. The main characteristics of the above lesions are that they can cause headaches, optic disc edema, and severe intracranial pressure increase, but no space-occupying lesions are visible in the imaging examination, and there are no other local signs in the neurological examination. Cerebral dural sinus embolism (including large cerebral venous embolism) and benign intracranial hypertension are almost indistinguishable clinically. However, cerebral dural venous sinus embolism is acute, headache is mostly at the top, and epilepsy can occur. Attention to the shape of the superior sagittal sinus on MRI or contrast-enhanced CT is helpful for differential diagnosis.

The diagnostic criteria are as follows:

1. There are symptoms and signs of increased intracranial pressure.

2. Nerve examination showed no localized signs.

3. Except for the increase of cerebrospinal fluid pressure, there is no abnormality in neuro-diagnostic examination (no deformation, displacement or obstruction of ventricle system).

4. The patient is conscious.

5. There are no other causes that can cause increased intracranial pressure.

6. If the cerebrospinal fluid examination is abnormal, the diagnosis is unsuccessful.

For benign intracranial pressure increase and congenital abnormalities, it is mainly timely diagnosis and early treatment.