What is dystrophic calcification?
Dystrophic calcification is a cellular reaction to a severely damaged or dead tissue. Large cells called macrophages begin with digest necrotic tissue. During this digestive process, calcium salt accumulates in the area, causing calcification of necrotic tissue. Calcification of degenerated tissue can occur anywhere in the body caused by autoimmune disease, infection and some cancer. Injured tissue can also develop calcified areas. Tissue inflammation occurs when the immune system begins to attack the body and as soon as it is attacked and damaged, these areas often develop cartilage or hyalinized scar tissue. Calcium deposits can develop when macrophages initially attack tissues or when white blood cells try to enjoy the scar. Healthcare providers prevent the calcification process in these disorders by disrupting the immune response. Treatment of these disorders often involves anti -inflammatory or immune drugs for prescription anti -inflammatory or immune suppression. The person who's withdrawalThey take parasitic or tuberculosis, regularly develop calcified areas around infected tissue when the immune system attempts to destroy foreign organisms. Treatment with the right medication generally eliminates infection and prevents further calcification. As sarcoma tumors begin to form, malignant cells often produce dystrophic formations of calcification. These calcified areas are often evident when bone sarcomas metastasize to close soft or distant breast tissues.
After an injury or traumatized tissue, blood can probed, resulting in bruising hematoma. Open wounds usually develop scars tissue. Each of these scenarios may result in dystrophic calcification resulting from a state known as traumatic osificans. Injection granulomas may occur when drugs are unintentionally injected into oily tissue, causing necrosis and scar formation. Cell infiltration can lead to sessionCalcium and treatment of granuloma or myositis may require surgical removal.
venous insufficiency, prolonged immobility and other conditions can produce blood clots, and these clots can calcify, causing Flebolith. Pleborlithy may occur mainly in organs located in the pelvic area and can form anywhere in venous structures. The arterial hardening begins as lipoprotein or oily bearings between the most inner and medium walls of the artery. Fagocytic cells normally spend these deposits, but when these cells die, they release chemicals attracting multiple phagocytes, causing inflammation, calcium deposition and plaque formation.