What Is Hand Atrophy?

Causes: First, part of the nerves of the hand or wrist are injured, second, the hand and wrist are fractured or ligament damage caused by long-term plaster or clip fixation, resulting in restricted movement. Another part is limited limb movement after hemiplegia, Anterior horn lesions, nerve roots, plexus, peripheral nerve lesions, abnormal immune function, genetic factors, heavy metal poisoning, nutritional metabolism disorders, and environmental factors can cause conduction disturbances of nerve excitement, thus causing the use of some muscle fibers. .

Hand muscle atrophy

Hand muscle atrophy is a type of muscle atrophy. Muscle atrophy refers to muscle contraction caused by striated muscle dystrophy, thinning or even disappearance of muscle fibers. Often the small muscles of one or both hands are atrophied and weak, and the forearm, upper arm, and scapular muscles are gradually involved. There are also those who begin with atrophy of the lower limbs, but it is rare, with significant distal atrophy, reduced muscle tone and tendon reflexes, no sensory disturbance, and sphincter function Involvement, involvement of the medulla oblongata, survival time period, often die of lung infection

Affected area

Limbs

Related diseases

Stroke fracture Shoulder-hand syndrome Open hand trauma Hand tendon injury Stroke sequelae

Related symptoms

Cachexia muscle atrophy spinal cord disease paralysis hand muscle atrophy weakness malnutrition myasthenia gravis

Affiliated Department

Department of Rehabilitation

Related inspections

Parathyroid hormone (PTH) electromyogram

Causes: First, part of the nerves of the hand or wrist are injured, second, the hand and wrist are fractured or ligament damage caused by long-term plaster or clip fixation, resulting in restricted movement. Another part is limited limb movement after hemiplegia, Anterior horn lesions, nerve roots, plexus, peripheral nerve lesions, abnormal immune function, genetic factors, heavy metal poisoning, nutritional metabolism disorders, and environmental factors can cause conduction disturbances of nerve excitement, thus causing the use of some muscle fibers. .

Diagnosis: EMG shows typical neurogenic changes. In resting state, fibrillation potential, positive sharp sowing, and sometimes beam tremor potential can be seen; motor unit potential duration widens, wave amplitude increases, polyphase wave increases during small force contraction, and vigorous contraction shows simple phase. Nerve conduction. Motor evoked potentials help determine upper motor neuron damage.

1. Mild atrophy: The muscle fibers are slightly decreased, the appearance of muscle tissue is not markedly sunken, the muscle tissue is loose when touched, the muscle is weak, and it can do resistance exercise.

2. Moderate muscle atrophy: Part of the muscle fibers atrophy and loss, the appearance of muscle tissue is sunken, the touch shrinks longitudinally, the transverse decreases, the muscle weakness is obvious, and can not do resistance exercise.

3. Severe muscular atrophy: Most of the muscle fiber tissue is atrophied and the related bones are exposed. Only a small amount of muscle fibers remain in the muscle tissue, and the muscle weakness is severe. The patient loses the most basic ability to coordinate movement.

4. Complete atrophy: The muscle fiber tissue is completely atrophic, and the motor functions associated with its muscles are completely lost.

Differential diagnosis of hand muscle atrophy:

1, neurogenic muscle atrophy: mainly caused by spinal cord and inferior motor neuron disease. Found in spine vertebra osteogenesis, intervertebral disc disease, spinal nerve tumors, arachnoiditis, neuritis, plexus lesions, spinal nerve tumors, arachnoiditis, neuritis, plexus lesions, nerve damage, syringomyelia, motor neuron disease Guillain-Barre Syndrome, Brain Disorders and Spinal Cord Disorders.

2. Myogenic muscular atrophy: common in muscular dystrophy, dystrophic myotonia, periodic paralysis, polymyositis, trauma such as crush syndrome, ischemic myopathy, metabolic myopathy, endocrine Myopathy, drug-induced myopathy, neuromuscular transmission disorder myopathy such as myasthenia gravis, etc.

3. Disuse muscle atrophy: Upper motor neuron disease is caused by long-term inactivity of muscles, and systemic wasting diseases such as hyperthyroidism, malignant tumors, and autoimmune diseases.

4. Other causes of muscular atrophy: such as cachexia muscular atrophy, sympathetic muscular dystrophy, etc.

diagnosis:

1. Mild atrophy: The muscle fibers are slightly decreased, the appearance of muscle tissue is not markedly sunken, the muscle tissue is loose when touched, the muscle is weak, and it can do resistance exercise.

2. Moderate muscle atrophy: Part of the muscle fibers atrophy and loss, the appearance of muscle tissue is sunken, the touch shrinks longitudinally, the transverse decreases, the muscle weakness is obvious, and can not do resistance exercise.

3. Severe muscular atrophy: Most of the muscle fiber tissue is atrophied and the related bones are exposed. Only a small amount of muscle fibers remain in the muscle tissue, and the muscle weakness is severe. The patient loses the most basic ability to coordinate movement.

4. Complete atrophy: The muscle fiber tissue is completely atrophic, and the motor functions associated with its muscles are completely lost.

Prevention: Nerve nutrition drugs and blood vessel drugs can be used.

First, maintain an optimistic and happy mood. Stronger long-term or repetitive emotional changes such as nervousness, anxiety, irritability, and pessimism can make the cerebral cortex excite and inhibit the balance of the process of imbalance, increase muscle jumps, and develop muscle atrophy.

Secondly, rationally adjust the diet structure. Muscular atrophy patients need high-protein, high-energy dietary supplements, which provide the necessary substances for the reconstruction of nerve cells and skeletal muscle cells to strengthen muscle strength and muscle growth. Early use of high-protein, rich in vitamins, phospholipids And trace element foods, and actively cooperate with medicated diets, such as yam, barley, lotus seed heart, tangerine peel, ginseng, lily, etc., fasting spicy foods, quit smoking, alcohol. Middle and advanced patients with high protein, high nutrition, rich Energetic semi-liquid and liquid foods are mainly used, and small meals and frequent meals are used to maintain the nutrition and water-electrolyte balance of patients.

Third, the combination of work and rest. Forcible sexual function training is forbidden, because forced sexual function training will be skeletal muscle fatigue, which is not conducive to the recovery of skeletal muscle function, regeneration and repair of muscle cells.

Fourth, strictly prevent colds and gastroenteritis. Due to low autoimmune function or some kind of immune deficiency in patients with muscular atrophy, once the muscular atrophy suffers from a cold, the disease becomes worse, the course of disease is prolonged, muscle atrophy, muscle jumps become worse, especially Patients with bulbar palsy are susceptible to lung infections.If not treated in time, the prognosis is poor, and even the patient's life is endangered.