What is Radiculopathy?

Radiculopathies are a class of diseases caused by disc herniation with or without spinal degenerative changes. It manifests as pain and segmental neurological disorders. The lesions of the anterior root (motor root) cause muscle strength and muscle atrophy dominated by the nerve root, and the lesions of the posterior root (sensory root) cause symptoms such as sensory disturbance in the corresponding dermal region. It can be treated with medicine, and severe symptoms require surgery.

Nerve root disease

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Radiculopathies are caused by

Nerve root dysfunction is usually secondary to chronic compression or invasion of the nerve root. The characteristic nerve root syndrome is characterized by pain and segmental neurological disorders. The lesion of the anterior root (motor root) causes the nerve root to innervate. Muscle weakness and muscle atrophy. Lesions of the posterior root (sensory root) cause sensory disturbance in the corresponding dermal region. Tendon reflexes corresponding to the nerve root segment weaken or disappear. Occupancy at the neck, chest, and waist levels is large Lesions, especially in patients with spinal stenosis, can cause compression on both the nerve root and the spinal cord. In addition to the nerve root syndrome, it can also increase the symptoms of spinal cord disease.

Radical pain can be triggered by spinal activity, coughing, sneezing or Valsalva-type breath hold, the latter through

A plain radiograph of the spine can show signs of degenerative arthritis or metastatic disease. CT scans can determine the diameter of the spinal canal and whether the lateral crypts have been invaded. MRI can provide excellent imaging of the spinal and spinal canal lesions In many cases, it has replaced spinal angiography. Cerebrospinal fluid cytology, protein, sugar and culture tests can distinguish cancerous meningitis from other chronic meningitis. Electromyography and evoked potential tests can help determine nerve roots Level and severity of the lesion. Nerve conduction velocity is normal, but in muscles innervated by one or more damaged nerve roots, spontaneous potential activity (normal phase wave and fibrillation) and action at maximum muscle contraction can be observed Attenuation of potential recruitment patterns. After repeated stimulation of the median, peroneal, or posterior tibial nerves, delays in evoked potentials at the level of the nerve root entry zone can be observed.

Muscle relaxants, analgesics, percutaneous electrical stimulation of the nerves or various local treatments can provide symptomatic relief of symptoms. Special treatments must address the etiology of radiculopathy. For epidural tumors or In the case of meningiomas, radiation therapy can be performed on the corresponding spinal cord segment, with adrenocortical hormones (dexamethasone 10 mg intravenously as a booster dose, and then 4 mg every 6 hours). If symptoms and signs continue to progress rapidly, you may need to Decompression by surgery. Surgical treatment of spinal cord compression caused by bone deformity is usually required. In cases of diffuse cancer of the brain (spinal membrane), intrathecal injection of methotrexate can be used to delay the progression of cancer Epidural and subdural abscesses require immediate surgical drainage for 6 to 8 weeks of antibiotic treatment. Treatment of symptoms caused by diabetes is effective in controlling blood sugar. Severe shingles infections need to be treated with antiviral drugs (Such as acyclovir, or arabinosyl adenosine). Early administration of adrenal corticosteroids in patients over 60 years of age may reduce the incidence of post-herpetic neuralgia. Fibroids require surgical removal.