What Is Ashman Phenomenon?
The Ashman phenomenon refers to the myocardial conduction tissue refractory period is affected by the heart rate, that is, the faster the heart rate, the shorter the refractory period of the ventricular atrial muscle, the longer the refractory period of the atrioventricular node, and vice versa
Ashman phenomenon
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- TCM disease name
- Ashman phenomenon
- English name
- Ashman
- The Ashman phenomenon refers to the myocardial conduction tissue refractory period is affected by the heart rate, that is, the faster the heart rate, the shorter the refractory period of the ventricular atrial muscle, the longer the refractory period of the atrioventricular node, and vice versa
- As early as 1910, Lewis discovered that intraventricular conduction abnormalities may occur when supraventricular arousal and ventricular arrhythmia occur, causing the QRS waves of ventricular depolarization to become large deformities. However, for a long time afterwards, the QRS waves of the ECG were still regarded as ventricular rhythms. In 1947, Gouaux and Ashman discovered that a premature heart beat after a long RR interval was often accompanied by QRS aberrations. After this, this special kind of electrocardiogram phenomenon is called Ashman phenomenon (Ashman phenomenon).
- 1 indoor differential conduction
- When the Ashman phenomenon occurs, the precardiac cycle is relatively long, which will cause the ventricular muscle refractory period to prolong in the next cardiac cycle. As a result, when supraventricular agitation is transmitted to the ventricle again, it can cause abnormal ventricular conduction. Ventricular depolarization of the QRS wave appears differentially transmitted indoor, which can be distinguished from the deformed QRS wave caused by ventricular premature beats.
- 2 Venturi phenomenon
- The principle of Wen's phenomenon is that the pathological prolongation of the absolute refractory period and the relative refractory period of the atrioventricular conduction tissue is mainly prolonged by the relative refractory period. Excitation cannot be conducted during the absolute refractory period, and diminished conduction occurs during the relative refractory period, and the conduction speed is slowed down. At the beginning of the Venturi cycle, the first supraventricular agitation falls into the final phase of the atrioventricular conduction tissue response or refractory period, and the PR interval is normal or slightly prolonged. According to the Ashman phenomenon, the refractory period of the ventricular muscle and the atrial muscle is shortened, the refractory period of the atrioventricular node is prolonged, and the second relatively refractory period that is excited into the atrioventricular conduction tissue, the PR interval is prolonged, and the ventricular beat is delayed. According to the Ashman phenomenon, since the second stroke, the atrioventricular conduction time is gradually extended, but the increase is gradually decreasing, and the decreasing conduction is more obvious. The last time in the Venturi cycle, which fell into the atrium of the atrioventricular conduction tissue, was blocked and blocked, and a QRS complex leak occurred, and a long RR interval occurred. After this long interval of atrioventricular conduction tissue, excitability recovers and a new cyclical change begins. When the cardiac cycle changes, the occult conduction, and the vagus nerve tension fluctuate, the gradual extension of the PR interval will also change, forming an atypical Venturi phenomenon.