What are different treatments for HIV?
most people compare to receiving news that HIV is positive with the infection of fatal illness. But HIV is not deadly in itself. The HIV mechanism is classified as retrovirus and disrupts the immune system to the extent that the body cannot defend itself from other threats that can move from something as aggressive as cancer to otherwise relatively benign infection. The virus does this by transforming its own RNA ribonucleic acid (RNA) to deoxyribonucleic acid (DNA) using an enzyme known as a rNA-reverse transcriptase as Vehiculum. This newly created DNA then replaces the normal DNA of the infected cell to replicate itself. Other auxiliary lymphocytes, such as CD4 cells and their subgroups that carry the T4 marker, begin to multiply in response to this invasion. However, these cells also become HIV. This process continues until the number of lymphocytes is destroyed and replaced by HIV DNA cells. At this point the infected person arrived in the late phase of Hi infectionIn or AIDS.
Since the functioning of the immune system is thus critical in fighting HIV infection and slowing its progression to AIDS, the objective of HIV treatment begins with optimal nutritional support. This means that a HIV positive individual should strive to achieve a balanced and healthy diet that has high grains, seeds, nuts, fruits and vegetables and low fats and refined flour and sugars.
Nutritional treatment of HIV should also include antioxidant therapy. Several studies have found that individuals infected HIV have a deteriorated antioxidant defense system, either because of poor nutrition or inability to absorb nutrients. This condition allows increased peroxidation of lipids (fat disintegration) that may promo promo promotions of HIV. The aim is that the aim is to increase the levels of GluthatHione amino acid in white blood cells by increasing antioxidant activity.
should also be considered a specific shoeAnical treatment of HIV. For example, epigallocatechin-3-galt (EGCG), the active ingredient of green tea, has shown a strong ability to prevent HIV replication when introduced by infected lymphocytes. The olive leaf extract also shows viral activity of anti-HIV by preventing the replication and expression of P24 in infected cells. Scientists suspect that the active ingredient of the olive leaf, oleuropeine, which is converted into olenic acid in the body, may be behind the mechanism of inhibition of viral replication.
One important note: although Echinacea herb is introduced as a strong immuno-modulation therapy, patients infected HIV should take it not . The reason is to stimulate the production of T-cells for the herb, which could help help HIV in the transfer of its DNA to these cells and the replication itself.
More aggressive HIV treatment includes antiretroviral medicines. There are several classes of these drugs, including protease inhibitors (PI), inhibitors of chemokin co-ceptors, nickl inhibitorsEoside analog reverse transcriptases (NRTI) and others. In this group, it was the first antiretroviral drug to be introduced, which works by suppressing the enzyme HIV, a rNA-reverse transcriptase. In the other hand, drugs are inhibited by another enzyme known as HIV protease. Chemokin-inhibitors are a relatively new HIV treatment that are designed to focus on a specific HIV infection known as the CCR5-tropic HIV-1.