What Is a Pulmonary Shunt?

Intrapulmonary shunt refers to the fact that intrapulmonary blood flow is directly mixed with oxygenated and arterialized blood without oxygenation for different reasons to reduce blood oxygenation. Its properties are similar to "right to left in patients with congenital heart disease" "Shunt", but it occurs in the lung, so it is intrapulmonary shunt, also known as venous blood doping. The blood from the normal bronchial vein and the smallest cardiac vein goes directly into the left heart without gas exchange, forming an intrapulmonary shunt, but the amount accounts for less than 2% of the cardiac output. In the change of the V / Q ratio imbalance, if the ventilation is less than the blood flow, it can cause different degrees of venous blood doping, or intrapulmonary shunt-like changes; if the ventilation is completely stopped and the blood flow continues, pathological lungs are formed Internal shunting, which is the most serious form of ventilation disorder. Airway obstruction, pneumonia, atelectasis, and pulmonary edema can cause intrapulmonary shunts if the blood flow in the capillaries cannot be exchanged with alveolar gas, that is, the blood flow fails to obtain oxygenation and arterialization. In severe pulmonary lesions, intrapulmonary shunts can account for as much as 30% to 50% of cardiac output. Patients develop severe hypoxemia and cyanosis, which cannot be corrected by ordinary oxygen inhalation.

Intrapulmonary shunt

Right!

Chinese name

Intrapulmonary shunt

as a result of

Mixing unoxygenated blood with oxygenated blood

Intrapulmonary shunt

30-50% of cardiac output in lesions

Attributes

Can not be corrected by ordinary oxygen inhalation

Intrapulmonary shunt refers to the fact that intrapulmonary blood flow is directly mixed with oxygenated and arterialized blood without oxygenation for different reasons to reduce blood oxygenation. Its properties are similar to "right to left in patients with congenital heart disease" "Shunt", but it occurs in the lung, so it is intrapulmonary shunt, also known as venous blood doping. The blood from the normal bronchial vein and the smallest cardiac vein goes directly into the left heart without gas exchange, forming an intrapulmonary shunt, but the amount accounts for less than 2% of the cardiac output. In the change of the V / Q ratio imbalance, if the ventilation is less than the blood flow, it can cause different degrees of venous blood doping, or intrapulmonary shunt-like changes; if the ventilation is completely stopped and the blood flow continues, pathological lungs are formed Internal shunting, which is the most serious form of ventilation disorder. Airway obstruction, pneumonia, atelectasis, and pulmonary edema can cause intrapulmonary shunts if the blood flow in the capillaries cannot be exchanged with alveolar gas, that is, the blood flow fails to obtain oxygenation and arterialization. In severe pulmonary lesions, intrapulmonary shunts can account for as much as 30% to 50% of cardiac output. Patients develop severe hypoxemia and cyanosis, which cannot be corrected by ordinary oxygen inhalation.