What is autoimmune thrombocytopenia?
Autoimmune thrombocytopenia (AITP) is a health condition in which the immune system of the body develops antibodies against its own plates, resulting in thrombocytopenia or a low number of inserts. This may be due to various conditions, including reactions to drugs, viral infections and other systemic diseases. In other cases, this disease develops people who are otherwise healthy. Symptoms may include easy bleeding and rashes. The treatment depends on the reason why it has evolved.
In general, autoimmune thrombocytopenia occurs when the body produces antibodies against its own plates, resulting in the destruction of the plates. These antibodies can be produced in many clinical situations. When some people use specific drugs, their bodies could respond against drugs and produce antibodies that damage the platelets of these people. Similarly, reactions to certain viral infections can also cause the production of these antibodies. In other cases antibodies are producedthe hits idiopathically or without a particular stimulating factor.
Symptoms of autoimmune thrombocytopenia may vary. If the disease is mild, patients may not have any symptoms and may be diagnosed only if laboratory studies performed for other reasons reveal an abnormal number of plates. Other patients could be symptomatic and experience easy bleeding of gums, nosebleeds and skin rashes. If the disease is severe, patients may develop internal bleeding that can be deadly.
Diagnosis of autoimmune thrombocytopenia can be complicated. One of the main features of the disease is the low number of plates. It is important that the physician distinguishes autoimmune thrombocytopenia from other causes of low platelet, so it is usually important that the physician causes a complete history of health problems, medications and any recent illnesses from any Pataient that BYlo found to have a low number of plates. Laboratory studies can also be conducted in attempts to identify some antibodies that are responsible for the destruction of the plate. Examination of peripheral blood under the microscope and, in some cases, exploring the aspired bone marrow sample can often also provide traces of diagnosis of this condition.
Treatment of autoimmune thrombocytopenia depends on the reason why antibodies have developed against plates. If the drug is caused, the drug should be stopped; If it is associated with a viral infection, basic infection should be treated. In idiopathic cases, depending on the severity of the disease, different treatment options are possible. Mild diseases do not require treatment. Severe diseases could be treated with therapies such as administration of immunoglobulin or plasmapheresis.