What Is Pulmonary Vascular Resistance?

Pulmonary hypertension caused by abnormal pulmonary blood vessels due to long-term congenital heart disease

Pulmonary vascular disease

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Pulmonary hypertension caused by abnormal pulmonary blood vessels due to long-term congenital heart disease

Pulmonary vascular disease may be a limiting factor in caring for babies and children with congenital defects (such as ventricular septal defect, atrioventricular channel defect, permanent arterial trunk), high pressure shunts from pulmonary vascular disease (ventricle, aorta-lung) and Pulmonary hypertension exists alone, which maintains a high level of pulmonary vascular resistance, with pulmonary hypertrophy and obstruction of many branch lumens.

Western Medicine Name

Pulmonary vascular disease

Disease site

Pulmonary vessels

Main cause

Pulmonary hypertension caused by abnormal pulmonary blood vessels due to long-term congenital heart disease

Multiple groups

Babies and children

When the pulmonary vascular resistance reaches and is equal to the resistance of the systemic circulation, the left-to-right shunt is reduced, while the right-to-left shunt results in a decrease in systemic blood oxygen saturation and a visible cyanosis. The continuous and gradually increasing right-to-left shunt leads to the periphery Hypoxia and excessive production of blood cells, the increase in oxygen capacity is manifested as a hematocrit of more than 65%, but when the hematocrit is high, the increase in blood cell viscosity causes the release of oxygen into the tissue.

When the calculated pulmonary vascular resistance is greater than 1/2 of the systemic circulation resistance, it is often a contraindication for surgery. If the pulmonary vascular resistance is higher than normal, the evaluation before surgery needs to be very careful. For patients with Eisenmenger symptoms (If there are too many blood cells, visual problems, and increased fatigue) it is beneficial to carefully perform venous bleeding and maintain total blood volume, reducing the hematocrit level from> 65% to about 60%.

Although studies using drugs such as cycloprostaglandin are ongoing, there is no specific treatment for existing pulmonary vascular diseases.