What is the intestinal metaplasia?

intestinal metaplasia generally involves the development of intestine, such as cells, in places where this type of cell is usually not found. The condition is usually attributed to chronic inflammation caused by bacteria, person or environmental factors. The differentiated cell type of cells often develop in the stomach and esophageal area, but can occur anywhere in the body. Treatment may include the removal of causal factors.

The state generally occurs when the constant regeneration process changes normal spinocellular cells to columnar mucosal cells. Cell membranes and nuclei are usually distorted and have vacuoles that contain sour mucin. As the name suggests, transformed cells resemble cells found in the intestines. These abnormal cells are joined and may spread in the affected area. Although it is generally considered to be a benign change, the condition is often compared to pre-alignic.

Focal intestinal metaplasia is one of two classifications of fitness and has two variations. This focal form usually occurs inreaction to chronic injury or mucosal irritation and is further classified as complete or incomplete. Complete focal forms of the state generally produce cells that resemble those commonly found in the small intestine. Cells are subject to the ripening process that allows them to appear and function as intestinal cells. They can eliminate peptides and absorb nutrients.

The second variation of focal metaplasia is commonly referred to as incomplete intestinal metaplasia. In this form, transformed cells are similar to microscopic structures found in the large intestine. The affected organ generally contains cup cells and basic villas, often associated with the large intestine.

The

GLAND Hall of intestinal metaplasia usually develops secondary to ulcerative conditions. It is the second main classification of intestinal intestinal. The erosion of the stomach glands may eventually retreat to the development of the mucous membranes.

doctors may evaluated during endoscopicIt discovers intestinal metaplasia. The initial phase of the state often occurs as white plaques or spots. Abnormal cells can also occur as a colored area. Definitive diagnosis usually includes biopsy and microscopic examination. In certain circumstances, the condition may be reversible.

Pathological stress caused by Helicobacter pylori often induces inflammation and ulceration of tissue, which can lead to intestinal metaplasia. Antibiotic treatment regimens can usually eliminate bacteria and subsequent irritation. Physiological stress factors that contribute to stomach irritation include diet with vitamin C deficiency, alcohol consumption and smoking. The condition can also create chronic use of non -steroidal anti -inflammatory drugs.

Some autoimmune disease attacks the parietal and the main cells of the stomach, creating chronic inflammation and injuries. In many cases, the reversal of controllable tissue factors allows the opportunity to normally heal. Individuals who have a rangeRry or serious cases of intestinal metaplasia may undergo ablation or surgical removal of damaged tissue.

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