What is endometrial hyperplasia?

endometrial hyperplasia is a condition in which cells of endometrium, uterine lining, grow faster than normal. There are two forms of endometrial hyperplasia: typical and atypical. In a typical form of the uterus cell, no changes have undergone due to the endometrial state. In atypical form of hyperplasia, cells have undergone changes that could develop on uterine cancer if they were not treated.

During the menstrual cycle, the endometrium intensifies in preparation for pregnancy and becomes a layer of cells and blood -rich blood. If pregnancy does not occur during this cycle, the endometrium is shed as a menstrual period. Endometrial thickening is controlled by estrogen and progesterone. If estrogen production is abnormally high or the production of progesterone is unusually low, the endometrium will grow into a changed estrogen ratio to progesterone. This overgrowth is called Hyperplasia.

Any condition that changes the estrogen to progesterone can increase the risk of hyperplAzie. Therefore, endometrial hyperplasia occurs more often in women who are in menopause or near or in their vicinity or which have menstrual cycle disorders such as irregular periods. Diseases such as diabetes or polycystic ovary syndrome may also increase the risk of endometrial hyperplasia. In addition, menopausal women who only use substitution therapy are exposed to greater risk of endometrial hyperplasia.

The main symptom of endometrial hyperplasia is abnormal menstruation. A woman with this condition often has heavy or irregular periods and can also bleed during the menstrual cycle. Menstruation can also be unusually painful. The diagnosis of endometrial hyperplasia is usually done on the basis of these symptoms and test results, such as ultrasonund or hysteroscopy that allows doctors to view the interior of the uterus and take endometrial tissue samples.

uWomen with typical hyperplasia may include endometrial therapy of hyperplasia to include hormonal therapy to provide further progesterone. This helps normalize the ratio of estrogen to progesterone, which prevents the overgrowth of the endometrium. Women with atypical hyperplasia have a significantly increased risk of uterine cancer and therefore have to consider more radical treatment. The most common option is hysterectomy, but women who wish to have children in the future could prefer other options. A common alternative to hysterectomy is a stronger version of hormonal therapy used for women with typical hyperplasia.