What is intimimal hyperplasia?

intimal hyperplasia is an exaggerated type of healing that occurs in the lining of blood vessels. This is a problem because it causes thickening the lining of the blood vessels that can narrow the inside of the blood vessel and prevent normal blood flow. Intimal hyperplasia usually occurs in conjunction with some forms of blood vessel surgery, procedures where the vessels are expanded using balloons and techniques where tubes are inserted to maintain open containers. Sometimes, a bypass graft can be affected by hyperplasia, where the blood vessel from another place is used to redirect the flow around the blocking. There is no truly effective treatment and research is in solving this problem. The hyperplasia process is usually triggered by damage to the lining. This can be caused by a procedure such as endarterectomy, where the accumulation that causes tapered arteries is cleaned. White blood cells, plates and other substances move to the damaged area and release special substances that affect muscle cells in the wall of the blood vessels. Muscle cells RosThis into the intima tunica and creates a solid scar that thickens the lining and narrows the blood vessel.

Although intimal hyperplasia causes problems, it is a really extreme form of a normal process that is constantly inside the blood vessels. The development of intimal hyperplasia is a special problem in the treatment of coronary arteries, where the balloons are inflated inside the narrowed arteries to expand them, and the tubes called the stents are inserted to maintain extended arteries open. The inflation procedure and stent insertion have the potential to damage the arterial lining and cause intimal hyperplasia. If hyperplasia occurs, it usually develops a month to a year after the procedure.

doctors seek to prevent intimimal hyperplasia by ensuring that they do not harm the lining of blood vessels during inflation or stent insertion. Were made specially coated stents to preventILO extension of hyperplasia inside the opening of the stent, but at each end of the tube there may be densification. Different medicines have been developed and further possibilities are investigated to inhibit intimal hyperplasia. Traditional medicines to attempt to fight the blood are also used to attempt to fight the problem.