What is Tophaceous DNA?

GOUT is a disease that causes sudden joint pain due to inflammation. The primary symptom of DNA is the elevated level of uric acid in the bloodstream. The main causes of this condition are diet rich in amino acid called purine or defect in metabolism of uric acid. Tophaceous DNA, also called chronic DNA, occurs in people with long -term increased levels of uric acid, leading to the formation of large crystals of uric acid in joints. Tophaceous DNA occurs as deposits of monosodium urate bearings, also called tophi, size enlargement. This usually occurs after several years of elevated uric acid levels and minor symptoms of DNA.

The primary symptom of DNA is severe joint pain. The pain is caused by the crystals that are formed inside the joint, which makes movement painful, as if the inflammation that occurs around the joint. The joint is often so tender that even a very mild touch can cause very serious pain. Among other possible symptoms of pAtti low fever, swelling, redness and stiffness in the affected joints. DNA most often affects the large foot of the legs, but it can also affect the ankle, heel or legs or joints in the knee, elbows, wrists, fingers or spine.

In people with tophaceous gout, symptoms increase severity when the settlement of the monosodial urate increases. As the deposits increase, they begin to expand outside the joint, causing noticeable lumps under the skin. In advanced cases, tophi can become so big that it breaks the skin. Tophi is visible as white chalk nodes surrounded by reddish and inflamed skin.

Tophaceous DNA complications can become serious if the nodes are not treated. DNA ITRELF, as well as the development of TOPHI, can seriously reduce mobility due to pain and stiffness of joints. In addition, when tophi breaks the skin surface, there is a risk of being infected or ulcerated. Infection can lead to potentiallya fatal disease called septicemia in which bacteria enter the bloodstream.

Tophaceous DNA treatment includes anti -inflammatory drugs and medicines that help to cope with pain and reduce uric acid levels. If uric acid levels can be reduced to a permanently low level, the crystallized monosodium urate eventually begins to dissolve, reducing the size of tophi knots and helping to alleviate symptoms. Dietary changes are also useful, including reducing high -purine food intake. If these treatments are not enough to stop or reverse the progress of the disease, surgery may be required.