What are synaptic vesicles?
Synaptic vesicles are structures found in the human body responsible for storing neurotransmitters. Neurotransmitters are essentially chemicals designed to carry signals from neurons to other cells in the body. This is done through synapses or intersections that provide signals pass. These synaptic vesicles are also known as neurotransmitter vesicles.
The vesicles are often described as a bubble made of liquid sitting inside another liquid. More precisely, it is a small bag surrounded by a membrane whose function is to store or carry other substances. In the case of synaptic vesicles, these substances are known as neurotransmitters.
The thin membrane of the synaptic vesicles is called the lipid double -layer. This membrane consists of two separate lipid molecules. Almost every living organism contains cell membranes that are made of this lipid double -layer. This membrane is responsible for ensuring that molecules such as proteins and ions, Remantheir right place.
Inserted into each neurotransmitter are transport proteins. These proteins are specific to the individual type of neurotransmitter. The function of these proteins is to help the substance or substance when the membrane is exceeded.
Synaptic vesicles are sometimes destroyed by substances known as neurotoxins. Neurotoxin is essentially a poison that affects nerve cells, also referred to as neurons. Botulinum toxins and tetanus toxins are among the neurotoxins known to destroy synaptic vesicles.
abnormalities in synaptic vesicles may also occur, as in health known as familial infantile myasthenia gravis. This disease is classified as an autosomal dominant disorder. This means that the disease is inherited and the defective gene can only come from one parent. Other types of genetic diseases require both parents to carry a deflectioniva gene.
In the familial infantile myasthenia gravis, synaptic vesicles are not properly developed. Infants experience such symptoms such as apnea or other breathing problems as well as fatigue and weakness. Feeding problems are also common with this disorder. Ophthalmoparesis or paralysis of the eye muscles is another common characteristic in children born with family infantile myasthenia gravis. Symptoms of this disorder very often begin to improve as soon as the child ages.
Treatment of familial infantile myasthenia gravis consists of using immunosuppressive drugs and cholinesterase inhibitors. If this approach is not successful, surgery is sometimes necessary. This procedure generally requires the removal of the gland of the thymus. The prognosis is generally favorable when many patients achieve normal life expectancy.