What Are Oncogenes?

An oncogene hypothrsis an hypothesis. It is believed that most or all vertebrate cells contain genetic information that determines the type C RNA tumor virus as a normal part of the genome, and that the virus-determining genes are usually in a repressed state. They are called viral genes (virogene), and some viral genes are called temple genes. When expressed, they determine changes in cell transformation. This hypothesis holds that in the presence of oncogenic factors, some or all of the viral genes are de-suppressed. According to The degree of viral gene de-repression, and the role of oncogenes can include the synthesis of viral particles and {or) the transformation of cells. The oncogene hypothesis is consistent with many observations. I (1) Some cells that did not show previous infection with the virus after repeated transplantation. Type C R11A tumor virus appeared "spontaneously" in cells; f2) In cells that had not previously been shown to contain the virus or its antigen (caused by oncogenic factors), the induction of mold was caused by (oncogenic factors), (3) the use of hybridization studies to prove that Non-synthetic type C viruses are cells that have genetic information for type C viruses. These observations are also consistent with the previral hypothesis} P} _ virus hypothesis}. ^[1]

Oncogene hypothesis

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An oncogene hypothrsis an hypothesis. It is believed that most or all vertebrate cells contain genetic information that determines the type C RNA tumor virus as a normal part of the genome, and that the virus-determining genes are usually in a repressed state. They are called viral genes (virogene), and some viral genes are called temple genes. When expressed, they determine changes in cell transformation. This hypothesis holds that in the presence of oncogenic factors, some or all of the viral genes are de-suppressed. According to The degree of viral gene de-repression, and the role of oncogenes can include the synthesis of viral particles and {or) the transformation of cells. The oncogene hypothesis is consistent with many observations. I (1) Some cells that did not show previous infection with the virus after repeated transplantation. Type C R11A tumor virus appeared "spontaneously" in cells; f2) In cells that had not previously been shown to contain the virus or its antigen (caused by oncogenic factors), the induction of mold was caused by (oncogenic factors), (3) the use of hybridization studies to prove that Non-synthetic type C viruses are cells that have genetic information for type C viruses. These observations are also consistent with the previral hypothesis} P} _ virus hypothesis}. ^[1]