What Is Aplasia Cutis?
At birth, there are clear-cut skin defects with rough bases and red granulomas, which appear as a large thick-walled bullae, and the top quickly falls off. The scalp defect is about 60%, usually 2cm, which can be as large as 9cm. It is long, triangular and star-shaped. Defective wounds heal slowly, can scab repeatedly, and fall off for months or even years. May be accompanied by other developmental abnormalities, such as congenital amputation, most patients with bullous epidermolysis.
Skin dysplasia
- Skin dysplasia (alplasia cutis), also known as congenital skin defect, is epidermal dermis in one or several areas, and sometimes even subcutaneous tissue has congenital defects.
Skin dysplasia signs and symptoms
- At birth, there are clear-cut skin defects with rough bases and red granulomas, which appear as a large thick-walled bullae, and the top quickly falls off. The scalp defect is about 60%, usually 2cm, up to 9cm, or It is long, triangular and star-shaped. Defective wounds heal slowly, can scab repeatedly, and fall off for months or even years. May be accompanied by other developmental abnormalities, such as congenital amputation, most patients with bullous epidermolysis.
Skin dysplasia medication
- Pay attention to care during infancy, control secondary infection, and later can be corrected by plastic surgery.
Skin dysplasia diet health care
- Rambutan
- Nile Redfish
Skin dysplasia prevention care
- Early detection and early treatment.
Pathogenesis of skin dysplasia
- It is autosomal dominant or recessive. It may also be related to primary differentiation defects and adjacent amnion developmental defects.
Diagnosis of skin dysplasia
- Sometimes it can be confused with forceps trauma, but it can be diagnosed from its shape, location and clear puncture edge. Epidermal moles should also be identified.
Skin dysplasia test method
- Laboratory inspection:
- Currently there are no related content description.
- Other auxiliary checks:
- Histopathology: Poor regeneration, varying in depth, may have epidermal and dermal structures missing, and fat in the subcutaneous tissue may be partially or completely missing. It is characteristic that the epidermis has no appendages.
Skin dysplasia complications
- May be accompanied by other developmental abnormalities, such as congenital amputation, most patients with bullous epidermolysis.
Prognosis of skin dysplasia
- Defective wounds heal slowly, can scab repeatedly, and fall off for months or even years. May be accompanied by other developmental abnormalities, such as congenital amputation, most patients with bullous epidermolysis.
Pathogenesis of skin dysplasia
- The pathogenesis is not yet clear. May be related to primary differentiation defects and adjacent amnion developmental defects.