What is secondary hypothyroidism?
Reduced thyroid activity resulting from the impaired function of the hypothalamus or pituitary gland is called secondary hypothyroidism. This form of hypothyroidism is often diagnosed in women aged 50 years and older, with adequate treatment. Secondary hypothyroidism is associated with serious complications, including heart disease, coma and death. The treatment of this condition is focused on restoring suitable hormone levels and proper thyroid function.
There are two classification of hypothyroidism: primary and secondary. Primary hypothyroidism is a disruption of the correct production of hormones that comes from the thyroid gland. Secondary hypothyroidism occurs when the thyroid gland is adversely affected by damage to the pituitary or hypothalamus. When the T3 and T4 production becomes dihypophysis and hypothalamus, which is limited, they increase hormone production to compensate for loss. Thyroid disorders such as hypothyroidism occur when the function of one of these organs inhibits. Results of secondary hypothyroidism when the road breaks upCE on the pituitary gland and hypothalamus and there is no release of the thyroid stimulating hormone (TSH) or the hormone of the release of thyrotropin (market).
Several situations can contribute to the development of secondary hypothyroidism. Individuals who have undergone radiation therapy or whose brain was otherwise exposed to radiation, may develop secondary hypothyroidism. Tumors that develop either in the pituitary or hypothalamus may also contribute to the development of hypothyroidism disorder. Those who develop inflammation or pituitary infections due to excessive blood or disease loss may be diagnosed with hypothyroidism.
There are several symptoms and symptoms of secondary hypothyroidism, which can directly and adversely affect the individual's ability to function. Over time, symptoms develop in severity and tend to gradually disrupt mental and physical abilities. An individual in the early stages of the development of hypothyroidismY may cause symptoms that include joint discomfort, weight gain and fatigue. Other symptoms may include hair thinning, physical weakness and cold intolerance. Signs that occur with the disorder procedure may include disruption of menstruation in women, excessive peeling or drying of the skin and speech or hearing disorder.
Physical examination performed in combination with blood and display testing can be used to confirm the diagnosis of hypothyroidism. The preliminary symptoms found during physical examination, which may indicate that the thyroid problem is the problem, are reduced heart rate, blood pressure and temperature. The display of display, including X -ray and magnetic resonance imaging (MRI), can be done to evaluate the state of the heart and pituitary. Other laboratory testing, as well as liver function, can be carried out to assess cholesterol and hormone levels.
Secondary treatment of hypothyroidism generally focuses on compensationRMON and restore the proper function of the thyroid gland. A prescription drug can be recommended for artificial hormone deficiency, which is a process known as hormone substitution therapy. In some cases, lifelong hormone substitution therapy may be required. Treatment of hypothyroidism caused by a tumor may require surgery to remove substitution therapy of matter and postoperative hormones.
The correct thyroid function can be restored with early diagnosis. Good prognosis depends on appropriate and continued treatment. Individuals with hypothyroidism who interrupt their hormone substitution therapy endangered by recurrence of symptoms and worsening symptoms.
complications associated with hypothyroidism may include infertility and heart disease. A potentially life -threatening complication of hypothyroidism is a rare state known as the coma of myxedema. Symptoms of this serious condition include unconsciousness, stressed breathing and reduced blood pressure. Individuals who show signs of suspicion of coma of myxedema,should look for immediate medical attention. Treatment of this complication includes intravenous administration of steroid drugs and substitution therapy.