How Do I Treat Stiff Neck Muscles?

This disease is a disease of muscular sclerosis, toe and thumb deformity caused by the progressive deposition of bone structure in muscle and connective tissue. The cause is unclear. Some of them are autosomal dominant. Some scholars report that there are four factors that cause ossification:

Swelling and hardening of neck muscles

Swelling and hardening of the neck muscles is a clinical symptom caused by ossifying myositis disease. Myositis ossificans (myositisossificans) is a disease of muscular sclerosis, toe and thumb deformity caused by the deposition of progressive bone structure in muscle and connective tissue. The cause is unclear. Some of them are autosomal dominant. Common in children or youth. Appears as abnormal congenital torticollis, torsion and swelling and hardening of neck muscles, but most of them without pain. All muscles can be involved. Vigorous exercise or trauma can cause muscle rupture, hemorrhage, and hematoma formation. Most patients absorb hematoma, but it can also cause muscle stiffness and ossification, and eventually cause stiffness and disability of the corresponding joints of affected muscles.

Affected area

neck

Related diseases

Cervical Spondylosis Cervical Spine Fracture Dislocation Cervical Spine Tuberculosis Ossifying Myositis Traumatic Ossifying Myositis Pediatric Progressive Ossifying Myositis Pediatric Cervical Fusion Syndrome Five Hard Swelling

Related symptoms

Cramp calcification Periosteum reaction Muscle atrophy Tension Neck muscle swelling Harden Neck Stiff Neck Tough cyst Fatigue Headache

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This disease is a disease of muscular sclerosis, toe and thumb deformity caused by the progressive deposition of bone structure in muscle and connective tissue. The cause is unclear. Some of them are autosomal dominant. Some scholars report that there are four factors that cause ossification:

Stimulating factors: Contusion usually accounts for 60% to 70%, which can lead to hematoma. This damage can be minor, with only a small amount of skeletal muscle or myofibrils being damaged.

Damage signal: The damaged tissue or cell secretes a signal protein.

Mesenchymal cells with defective gene expression, these cells can generate bone-like or cartilage-like cells after receiving appropriate signals.

There is an environment in which ossification occurs continuously, of which the signal gene is the most critical, and Urist named the gene BMP.

Diagnosis based on clinical symptoms.

The disease is common in children or young people. Appears as abnormal congenital torticollis, torsion and swelling and hardening of neck muscles, but most of them without pain. All muscles can be involved. According to clinical characteristics, the disease can be divided into stage :

1. Response period: rapid increase in mass, rapid calcification, and rapid swelling. Trauma is 1 to 2 months and can reach 4 to 10 cm.

2. Active phase: Active phase can be characterized by fever, high local skin temperature, tenderness, and hard mass.

3. Maturity stage: Shell-shaped osteochondrosis occurs in the mature stage, and growth stops during the recovery period. The hard mass usually becomes smaller after 1 year, and even disappears completely.

4. Recovery period.

The auxiliary examination methods for this disease are mainly X-ray examinations:

X-rays are characterized by limited swelling that can occur shortly after injury. 3-4 weeks after the injury, a dense hairy image is displayed within the swelling, and the adjacent bone will show a periosteal response. 6-8 weeks after the injury, the lesions were clearly surrounded by dense bone and had the appearance of new bone. The core of a soft tissue mass is sometimes cystic and gradually enlarges its lumen, and in the later stages, eggshell-like cysts appear. The mass contracted 5-6 months after the injury, so an x-ray translucent band appeared between the mass and the adjacent cortical and periosteum responses.

Differential diagnosis of neck muscle swelling and stiffness:

1. Neck stiffness: Neck stiffness refers to neck muscle tension, swelling, stiffness, cramps (cramps), etc., and neck movements are not flexible. Common in fatigue and cervical spondylosis. Neck stiffness is a continuous excessive muscle contraction, which not only reduces the blood supply to the neck muscles, but also causes the accumulation of metabolites such as lactic acid, which causes muscle ischemic pain. Pain in the back of the head and pain in the head may be tension headaches caused by head or cervical spine lesions.

2. Neck toughness: Neck movements are restricted. This symptom can be seen in cervical rib syndrome. Cervical rib syndrome is a complex clinical syndrome caused by important vascular and nerve compression in the thoracic outlet area. It is also known as cervical thoracic outlet syndrome, anterior oblique muscle syndrome, pectoralis minor syndrome, costal lock syndrome, excessive abduction. Syndrome, etc., refers to a series of upper limb vascular and neurological symptoms resulting from compression of the brachial plexus and subclavian arteriovenous veins at the exit of the thorax. The main clinical manifestations are pain, numbness in the shoulders, arms, and hands, even muscle atrophy, bruises in the hands, and purple radial arteries. Diagnosis based on clinical symptoms.

The disease is common in children or young people. Appears as abnormal congenital torticollis, torsion and swelling and hardening of neck muscles, but most of them without pain. All muscles can be involved. According to clinical characteristics, the disease can be divided into stage :

1. Response period: rapid increase in mass, rapid calcification, and rapid swelling. Trauma is 1 to 2 months and can reach 4 to 10 cm.

2. Active phase: Active phase can be characterized by fever, high local skin temperature, tenderness, and hard mass.

3. Maturity stage: Shell-shaped osteochondrosis occurs in the mature stage, and the growth stops during the recovery period. The hard mass usually becomes smaller after 1 year, and even disappears completely, which is self-limiting.

4. Recovery period.

The auxiliary examination methods for this disease are mainly X-ray examinations:

X-rays are characterized by limited swelling that can occur shortly after injury. 3-4 weeks after the injury, a dense hairy image is displayed within the swelling, and the adjacent bone will show a periosteal response. The edge of the lesion is clearly surrounded by dense bone, and has the appearance of new bone. The core of a soft tissue mass is sometimes cystic and gradually enlarges its lumen, and in the later stages, eggshell-like cysts appear. The mass contracted 5-6 months after the injury, so an x-ray translucent band appeared between the mass and the adjacent cortical and periosteum responses.

This disease is a complication of severe trauma. You can prevent its occurrence and development through efforts. Patients with joint dislocations and severe trauma should consult an orthopedist in time. Do not blindly massage or massage to cause secondary injuries. Joint dislocation should be reduced in time. If there is a large hematoma, puncture and puncture in time. Joint trauma should be braked early and fixed in functional position with plaster support. If there is swelling, the combination of traditional Chinese and western medicine should be used to make the swelling quickly subside. For patients with paraplegia caused by spinal cord injury, if a large hematoma of unknown origin is found in the lower extremity, puncture and puncture should be performed in time. These are effective methods to prevent secondary ossifying myositis after severe trauma.