What are the use for beta blockers in heart failure?

Beta blockers or beta-adrenergic blocking agents can be used to treat many harmful symptoms of heart failure. A disease that is a clinical condition that represents the inability of the heart to pump adequate amounts of oxygenated blood is routinely treated with drugs blocking beta. Beta blockers solve symptoms of heart failure caused by an excess of hormone class called catecholamines. In addition, beta blockers can treat other symptoms that may be present, including high blood pressure, chest pain and heart arrhythmia. The use of beta blockers in heart failure has shown to reduce the need for hospitalization, slow down the disease progression and reduce the overall risk of death.

The use of beta blockers in heart failure is primarily associated with the influence of a heart of heart rate. Treatment, through a sympathetic nerve system reduces the heart rate of the patient and prevents the heart from, andwould have to work harder because of the condition. However, this effect was not considered desirable for patients with heart failure when the drug was first studied. Reduced heart rate has the risk of deterioration of heart failure symptoms, but as the research continued, beta blockers have proved to be advantages that predominate over this risk. The exact etiology of a heart failure case is important if the doctor decides whether to use beta blockers. It seems that the case present due to the deteriorated filling of the chamber, unlike the case caused by impaired emptying of the chamber, responds better to beta blockers in heart failure.

In addition to their sympathetic effect on heart muscles, beta blockers affect the renin/angiotensin kidney system in heart failure. Betta blocking drugs cause hormone secretion, renina. As Renin decreases, the cascade of events is manifested that decreases the heart demand for oxygen. The cascade reduces the volume of extracellular fluid and increases the abilityBlood to hold and carry oxygen into the body tissue. Treatment of beta blocker can be complemented and in most cases supplemented with diuretics and inhibitors of the converting angiotensin (ACE) enzyme (ACE) that increase this effect.

Patients who have significant shortness of breath - dyspnoea - while remaining calm, are those who do not have to be candidates for beta with blockers. With severe shortness of breath, it can increase the risks associated with beta -blockers. Some patients are considered to be hemodynamically unstable if their blood bears oxygen well, even under normal circumstances; These patients may not be good candidates for treatment.

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