What is fat embolism?

fat embolism is a potentially fatal condition in which fat enters the bloodstream, usually due to physical trauma. This embolism occurs when adipose macro globule is introduced into the lungs. Fat can enter the circulatory system, and therefore the lungs, as a complication in surgery or other types of physical trauma, such as bone fracture or childbirth. Heavy burns, diabetes mellitus and pancreatitis can also lead to adipose embolism.

Fipline embolism is a type of pulmonary embolism. Lung embolism is defined as a blockage of arteries in the lungs caused by a substance transmitted by the bloodstream. Other forms of lung embolism include the embolism of blood clots, air embolism and amniotic fluid embolism. This explains why fat embolisms occur most often after fractures and operations that include bone cutting. The theory is that circulating free fatty acids, combined with shock, blood infection or blood loss that reduces the liver function, are toxic to the pulmonarytissue. Some authorities argue that this toxicity is the primary cause of fat embolism.

Usually embolism occurs in one isolated area due to dishonest blood. Examples of adipose embolism tend to be many and small. For this reason, different symptoms may be present.

Fipbolia syndrome (FES) sometimes results from adipose embolism. FES symptoms usually occur about 12-48 hours after injury. These symptoms may include shortness of breath, coma, delirium or rash. Occasionally, immediate and serious symptoms such as respiratory failure occur and can lead to death within a few hours. Unfortunately, the FES is fatal in about 70% to 90% of cases.

Although there is no guaranteed advantage, the risk of FES may be reduced by implementing certain measures. For example, the speed at which a fracture is treated can play a role in the risk of FES. Within this theory, rapid medical care can be obtained in obtaining a set fromLomenine to reduce the risk. The shortening of the time that the broken bone is able to move around means that the bone marrow is more likely to be inhibited by entering the circulatory system. It is assumed that the prophylaxis of steroids reduces the incidence of FES in the administration of high -risk patients.