What is a myocardial infarction that is not an el-el, myocardial infarction?
myocardial infarction without elevation (nstemi) is a type of heart attack that is caused by partial or almost full occlusion of coronary artery with thrombus or embolism. With a closed coronary artery, the heart muscle or myocardium becomes ischemic or insufficient in the supply of blood and oxygen, leading to cell damage and potential cellular death. Myocardial infarction, which is not elevation, can only be distinguished from unstable angina (UA) and st-elevation myocardial infarction with laboratory results. Compared to the stem, where the full thickness of the heart muscle supplied by a particular coronary artery is damaged, is a myocardial infarction that is a non -blood infarction (ACSONDOME), in the blood heart attack, it is only in the blood, which is a coronary ars. The ACS spectrum includes angina, UA and myocardial infarction. UA and non-ST-Eleinfarkt myocardium with a bathtub is difficult to distinguish from each other if there are no laboratory results, and sometimes connected together and marked as UA/Nstemi.
Stable angina is defined as discomfort of chest or arm that is connected and reproducible with stress and physical exertion. It is easier to rest for 5 to 10 minutes or by receiving sublingual nitroglycerin. Unstable angina is Angina, which occurs at rest or after more than 10 minutes, is serious or acute and lasts longer or more often than previously experienced episodes. Myocardial infarction that is not elevation is given as a diagnosis when the patient has symptoms of UA and evidence of death or necrosis of myocyte, as it is found by increasing serum biomarkers such as troponin and creatinine kinase-MB fraction (CK-MB).
Four factors contribute to the pathophysiology of UA/nstemi. The first is the rupture or erosion of the atherosclerotic plaque with the overlapping thrombus formation. The second is dynamic obstruction such as coronary artery spasm, and the third factor is progressive mechanical obstruction, usually due to the reinforced wall of blood vessels,As happens in atherosclerosis. The fourth factor is increased demand for oxygen or reduced oxygen supply, which happens in anemia or increased heart rate. Any of these processes can occur in combination in the development of the nst.
In the electrocardiogram (ECG), electrical activity of the heart is recorded. The ischemia in the nStemi is limited to a subendocardium, so the vector or direction of the ST segment is shifted to it and is usually considered to be the depression of the ST segment on the ECG. ECG should be done when one complains about chest pain and the doctor suspects the basic cardiovascular problem. Changes in the ST segment require immediate treatment.
treatment of UA/Nstemives use of anti-skeletons that expand blood vessels such as nitrates and beta-blockers. The thrombus lies includes using anticoagulant drugs such as heparin and anti -aggregation drugs such as aspirin. High -risk patients should undergo coronary angiography and revascularize coronary artery within 48 to72 hours. This is to prevent further myocardial damage and blood restoration in the heart.