What is an ischemic cascade?
ischemic cascade is a number of events that occur at the cellular level in the brain when the supply of oxygen is interrupted. It starts within a few seconds of an ischemic event, such as a stroke that blocks the blood flow to the brain area and can continue even after restoring the normal blood flow. Because the cascade contains a number of events that take place in a particular order, scientists believed that interventions could be possible during the ischemic event to stop the cascade and reduce neuron damage in the brain. This causes a number of events to increase the cellular calcium levels, the cell releases the amino acids to repair them, the calcium levels continue to peak, the cell wall breaks down and the organelles in the cell begin to die. This causes the cell itself to die, and when so, it releases toxins into the surrounding area, damaging adjacent cells, including cells with adequate oxygen supply.
How ischemic cascade continuedHe is and neurons die, the patient may begin to strive for neurological symptoms because the brain is no longer able to function normally. In addition, swelling in the brain can develop, put pressure on the brain and create potential for other medical complications. If the pressure does not get rid of, the damage may spread to other areas of the brain and the ischemic cascade may have far -reaching consequences.
ischemic cascade spills when people have strokes, heart attacks, respiratory arrests and other medical emergencies that limit oxygen supply to the brain. Neurologists have studied this process extensively to learn how it happens and what is involved in each phase, to potentially develop treatment for arresting cascade. They believe that the stopping of a series of chemical reactions could derail ischemic cascade, reduce neuron damage in the brain and reduce the risk that moves and other events will have long -term consequences.
An important aspect of ischemic cascade is the risk that it will continue to do soNew oxygen supply. The dead and dying cells in the brain will continue to release toxins as soon as they reach a point without a return. Patients experiencing ischemic events must be carefully monitored in order for the symptoms of complications to indicate further brain damage.