What is vasospastic angina?
vasospastic angina is a health condition in which the cramps of one or both coronary arteries reduce blood flow to the heart and cause angina or chest pain. Vasospastic angina differs from typical angina in that it usually occurs at rest. Prinzmetal's Angina and Angina variant are other names for the same condition. The term "angina" refers to both vasospastic and typical angina, whether they are stable or unstable.
After picking up oxygen in the lungs, blood flows to the heart with coronary arteries. When one of these arteries goes through a convulsion that significantly reduces blood flow, the heart muscles do not get oxygen and nutrients that need to be properly operation. This lack of oxygen leads to arrhythmias, palpitations and chest pain that may be serious. Patients with vasospastic angina generally get better than in patients with typical angina in treatment, but there is a certain risk of seriousarrhythmia or heart attack.
normally, Angina is expected to occur when the body is active, and includes a high amount of atherosclerosis - hardening of arteries caused by the accumulation of plaque made of cholesterol, fat and minerals found in the blood - in coronary arteries. Vasospastic angina is often recognized because of the regular occurrence of attacks that appear while the body is calm and even after a few hours of sleep. It is also characterized by the fact that the exercise test does not bring an attack. Coronary angiography could be performed in an effort to diagnose vasospastic angina. In some cases, a drug such as acetylcholine, for stimulating vasospasms and obtaining a definitive diagnosis is administered.
condition or individual attacks can be caused by an unusually high level of stress, excessive exposure to colds or certain medicines or medicines. It is known that cocaine, vasopressin and ergonovin lead to convulsions experienced in vasospastic angina. The exact biomechanical cause of the narrowing was not hundredsA novel, but a possible explanation, is that the cells in the arteries do not work. After releasing acetylcholine and performing its task of limiting the blood vessels, the endothelial cells in the lining of the arteries stimulate the nitric oxide, which relaxes or expands the blood vessels even more than they were narrowed. If the cells do not create sufficient nitric oxide, the narrowing will not act and follow angina.