What Is the Connection Between Alcohol and Dehydration?

The dehydrating agent can reduce the moisture in the material or remove the crystal water in the compound molecules.

Medicine or nursing dehydrating agent and industrial dehydrating agent are commonly used.

(1) Hypertonic dehydrating agents can improve

Many types, such as ethanol,

Dehydrating agent is taken away during combustion

Polyacrylamide (PAM) is water-soluble

Overview of Dehydrating Agents

In the acute phase of cerebrovascular disease, dehydrating agents were mostly used in patients with cerebral hemorrhage before the 1980s, and cerebral infarction was less used. After the 1990s, dehydrating agents have become the conventional medicine for bleeding or ischemic cerebrovascular disease. And some scholars believe that the excessive amount of dehydrating agent applied in the acute phase of cerebral hemorrhage, after a large amount of dehydration of the brain tissue, reduces the mechanical compression effect of the edema tissue on the bleeding focus, which can aggravate the bleeding. Recently, some scholars believe that although patients with cerebral infarction have increased intracranial pressure to varying degrees in the acute phase, most of them are relatively mild and can be balanced by the body's own metabolism. If the intracranial pressure is not high or light, the use of large doses of dehydrating agents severely disturbs the balance of normal intracranial pressure and brings inestimable consequences to rehabilitation. The main cause of patients with cerebral infarction in the acute phase is ischemia and hypoxia of the brain tissue and insufficient blood supply to the brain. Therefore, it is necessary to use it appropriately to receive more results with less effort. ^[3]

The role of dehydrating agent

1. Dehydrating agents are valuable for the control of cerebral hernia, which can help more than half of the patients with cerebrovascular disease to help through the threat of cerebral hernia and prolong the survival time of patients with cerebral hemorrhage.

2. The role of the dehydrating agent is to help the body to stop or reverse the development of the brain hernia or make it early when the body cannot rebalance the intracranial pressure through its own compensation mechanism and the development of the brain hernia occurs. Brain hernia is reset.

3. The dehydrating agent has no obvious effect on patients with cerebral hernia who are heavier, have a larger amount of bleeding, and develop the disease more rapidly.

4. The dehydrating agent is deep to the bleeding site, destroys the midline nerve structure and has been in a coma for a long time. The dehydrating agent with pulmonary infection is powerless.

5. The effect of mannitol is better in the dehydrating agent, and it is the medicine of choice in the acute phase of cerebrovascular disease. ^[3]

Indications for dehydrating agents

To properly master the use of dehydrating agents, you must first correctly determine the degree of increased intracranial pressure and whether the disease is progressively worse. The acute phase of cerebral hemorrhage can be distinguished from the following:

Hemiplegia or hemiparesis, consciousness, headache and vomiting are not severe. Although bloody lumbar puncture is seen, the CT report shows a small amount of bleeding, and the intracranial pressure is not too high (about 200mmHg or less), indicating that the amount of bleeding is not large. In this case, the patient's body is likely to be self-compensated. It is possible to give some 10% glucose without dehydrating agent, but closely observe the change of the condition. If the intracranial pressure is not too high or in the normal range, if strong force is given at this time Dehydrating agents, on the other hand, are self-defeating, artificially disturbing the balance of intracranial pressure, reducing the normal intracranial pressure, and increasing the complexity of future treatment.

The headache and vomiting are more severe or the condition gradually worsens during observation, and the paralysis is completely or lethargic. At this time, a half dehydrating agent 125ML can be given.

When coming to the clinic, a conscious coma or a coma appears during the observation. The CT indicates that the midline structure is affected. At this time, the patient's coma is due to the impact of cerebral edema compression on the relationship of the midline structure. A powerful dehydrating agent such as manna alcohol.

After the dehydrating agent is administered, within 1-2 hours of the strongest dehydration effect, the condition must be closely observed and compared with the condition before the medication in order to make an appropriate decision on the evolution of pathological changes. As a basis for gradually reducing the number and dosage of medications.

Within 5-6 hours after medication, the condition is stable and the signs do not increase, indicating that the progress of the lesion is not obvious. It should be combined with other preventive measures, and it should be administered every 6 hours and closely observed for 24 hours. This plan is for patients with cerebral hemorrhage. Regular usage. Usually within 3 days of bleeding, even if the lesion no longer expands, the edema around the lesion is present to varying degrees or slowly aggravates. At this time, carefully observe for 24 hours. In the process, no evidence of expansion of the lesion is found. When it can be said that the condition is stable, the dehydration medicine at this time should be maintained for 72 hours, and then gradually stop using it. However, during the discontinuation of the drug, when the dehydration drug is observed to rebound, the intermittent period of drug administration can be prolonged and the drug can be stopped slowly, which is reasonable. At this time, there are comorbidities such as pulmonary infections and changes in symptoms with deepening of consciousness, so the application time of dehydration drugs must be appropriately extended.

The condition worsens rapidly when coming to the clinic, and can be observed within 1-2 hours. At this time, an additional dehydrating agent should be used for observation. If the dehydrating agent is used twice in a row, the progress of the disease deterioration cannot be controlled, it means that Dehydrating agents cannot effectively relieve the pressure, and other measures must be considered to rescue them.

If the process of exacerbation of the disease appears slowly and only manifests in 5-6 hours, you can also increase the amount of dehydrating agent, or shorten the interval between administration, try with dexamethasone and other methods, but do a good job of surgical treatment Prepare to remove the hematoma before surgery to develop a hernia when the dehydrating agent still cannot control the deterioration.

If you are in the pre-cerebellar hernia or have cerebellar notch hernia when you come to the clinic, immediately push a strong dehydrating agent such as mannitol. If you receive a reversal of the symptoms or recover the cerebral hernia, continue to use the dehydrating agent every 4 hours to maintain it. If the symptoms do not improve after the medication or although the symptoms of cerebral hernia have not been completely eliminated and controlled, then you should quickly re-administer the dehydrating agent and prepare for surgical treatment. There are many patients in this category. The condition is more serious and the prognosis is poor. . ^[3]